Some new insights into the molecular mechanisms of pain perception

The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl– channels

(2010) Boyi Liu et al.   JOURNAL OF CLINICAL INVESTIGATION

Kv7 Channels as Targets for the Treatment of Pain

(2009) A. Wickenden et al.   CURRENT PHARMACEUTICAL DESIGN

Activation of TRPV1 in the spinal cord by oxidized linoleic acid metabolites contributes to inflammatory hyperalgesia

(2009) A. M. Patwardhan et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Excitation and sensitization of nociceptors by bradykinin: what do we know?

(2009) Kazue Mizumura et al.   EXPERIMENTAL BRAIN RESEARCH

Modulation of Chloride Homeostasis by Inflammatory Mediators in Dorsal Root Ganglion Neurons

(2008) Katharina Funk et al.   Molecular Pain

TMEM16A confers receptor-activated calcium-dependent chloride conductance

(2008) Young Duk Yang et al.   NATURE

Retigabine reduces the excitability of unmyelinated peripheral human axons

(2008) P LANG et al.   NEUROPHARMACOLOGY

Retigabine, the specific KCNQ channel opener, blocks ectopic discharges in axotomized sensory fibres

(2008) Carolina Roza et al.   PAIN

TMEM16A, A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity

(2008) A. Caputo et al.   SCIENCE