Resveratrol Attenuates Oxidized LDL-Evoked Lox-1 Signaling and Consequently Protects against Apoptotic Insults to Cerebrovascular Endothelial Cells

ACUTE KIDNEY INJURY CLASSIFICATION

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Resveratrol: Its Biologic Targets and Functional Activity

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Multiple molecular targets of resveratrol: Anti-carcinogenic mechanisms

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Effects of preconditioning on tight junction and cell adhesion of cerebral endothelial cells

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Runx2-mediatedbcl-2gene expression contributes to nitric oxide protection against hydrogen peroxide-induced osteoblast apoptosis

(2009) Wei-Pin Ho et al.   JOURNAL OF CELLULAR BIOCHEMISTRY

Resveratrol attenuates lipopolysaccharide-induced hepatitis in d-galactosamine sensitized rats: Role of nitric oxide synthase 2 and heme oxygenase-1

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Signal-transducing mechanisms of ketamine-caused inhibition of interleukin-1β gene expression in lipopolysaccharide-stimulated murine macrophage-like Raw 264.7 cells

(2009) Ta-Liang Chen et al.   TOXICOLOGY AND APPLIED PHARMACOLOGY

Lipopolysaccharide triggers macrophage activation of inflammatory cytokine expression, chemotaxis, phagocytosis, and oxidative ability via a toll-like receptor 4-dependent pathway: Validated by RNA interference

(2009) Tsu-Tuan Wu et al.   TOXICOLOGY LETTERS

Oxidation of LDL and its clinical implication

(2008) Eiji Matsuura et al.   AUTOIMMUNITY REVIEWS

Apoptotic insults to human HepG2 cells induced by S -(+)-ketamine occurs through activation of a Bax-mitochondria-caspase protease pathway

(2008) S.-T. Lee et al.   BRITISH JOURNAL OF ANAESTHESIA

Apoptotic insults to human chondrocytes induced by sodium nitroprusside are involved in sequential events, including cytoskeletal remodeling, phosphorylation of mitogen-activated protein kinase kinase kinase-1/c-Jun N-terminal kinase, and Bax-Mitochondria-Mediated caspase activation

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