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O-LinkedN-Acetylglucosamine Modification on CCAAT Enhancer-binding Protein β

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Hexosamine flux, the O-GlcNAc modification, and the development of insulin resistance in adipocytes

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Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity

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White Adipose Tissue as Endocrine Organ and Its Role in Obesity

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Survey of O-GlcNAc level variations in Xenopus laevis from oogenesis to early development

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Elevation of GlobalO-GlcNAc Levels in 3T3-L1 Adipocytes by Selective Inhibition ofO-GlcNAcase Does Not Induce Insulin Resistance

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