Endoplasmic reticulum stress in adipose tissue augments lipolysis

The Metabolic Syndrome and Chronic Liver Disease

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Survivors Versus Nonsurvivors Postburn

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The endoplasmic reticulum and junctional membrane communication during calcium signaling

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Open Questions in Lipid Droplet Biology

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Severe Injury Is Associated With Insulin Resistance, Endoplasmic Reticulum Stress Response, and Unfolded Protein Response

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Lipolysis Response to Endoplasmic Reticulum Stress in Adipose Cells

(2012) Jingna Deng et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Pharmacologic ER stress induces non-alcoholic steatohepatitis in an animal model

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Packaging of Fat: An Evolving Model of Lipid Droplet Assembly and Expansion

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Long-Term Persistance of the Pathophysiologic Response to Severe Burn Injury

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Direct binding of triglyceride to fat storage-inducing transmembrane proteins 1 and 2 is important for lipid droplet formation

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Not Just Fat: The Structure and Function of the Lipid Droplet

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Adipose Tissue Dysfunction Signals Progression of Hepatic Steatosis Towards Nonalcoholic Steatohepatitis in C57Bl/6 Mice

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The Portal Theory Supported by Venous Drainage-Selective Fat Transplantation

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Induction of Liver Steatosis and Lipid Droplet Formation in ATF6α-Knockout Mice Burdened with Pharmacological Endoplasmic Reticulum Stress

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POST-BURN HEPATIC INSULIN RESISTANCE IS ASSOCIATED WITH ENDOPLASMIC RETICULUM (ER) STRESS

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Asymmetrical dimethylarginine triggers lipolysis and inflammatory response via induction of endoplasmic reticulum stress in cultured adipocytes

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