Gliadin fragments promote migration of dendritic cells

Cutting Edge: IL-1 Controls the IL-23 Response Induced by Gliadin, the Etiologic Agent in Celiac Disease

(2014) K. M. Harris et al.   JOURNAL OF IMMUNOLOGY

Pivotal Advance: Bifidobacteria and Gram-negative bacteria differentially influence immune responses in the proinflammatory milieu of celiac disease

(2009) G. De Palma et al.   JOURNAL OF LEUKOCYTE BIOLOGY

Coeliac disease

(2009) Antonio Di Sabatino et al.   LANCET

Regulatory T-Cell Function Is Impaired in Celiac Disease

(2008) Marilena Granzotto et al.   DIGESTIVE DISEASES AND SCIENCES

Gliadin Induces an Increase in Intestinal Permeability and Zonulin Release by Binding to the Chemokine Receptor CXCR3

(2008) Karen M. Lammers et al.   GASTROENTEROLOGY

Mechanisms and Consequences of Dendritic Cell Migration

(2008) David Alvarez et al.   IMMUNITY

Mucosal tissue transglutaminase expression in celiac disease

(2008) Vincenzo Villanacci et al.   JOURNAL OF CELLULAR AND MOLECULAR MEDICINE

In Vitro Differentiation of Human Monocytes into Dendritic Cells by Peptic–Tryptic Digest of Gliadin Is Independent of Genetic Predisposition and the Presence of Celiac Disease

(2008) Maryam Rakhimova et al.   JOURNAL OF CLINICAL IMMUNOLOGY

Newly identified genetic risk variants for celiac disease related to the immune response

(2008) Karen A Hunt et al.   NATURE GENETICS

Detecting shared pathogenesis from the shared genetics of immune-related diseases

(2008) Alexandra Zhernakova et al.   NATURE REVIEWS GENETICS