Diabetic nephropathy is associated with increased urine excretion of proteases plasmin, prostasin and urokinase and activation of amiloride-sensitive current in collecting duct cells

Plasmin in urine from patients with type 2 diabetes and treatment-resistant hypertension activates ENaC in vitro

(2014) Kristian B. Buhl et al.   JOURNAL OF HYPERTENSION

The Epithelial Sodium Channel  -Subunit Is Processed Proteolytically in Human Kidney

(2014) R. M. Zachar et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC

(2013) René F. Andersen et al.   PEDIATRIC NEPHROLOGY

Validation of A&D TM-2430 upper-arm blood pressure monitor for ambulatory blood pressure monitoring in children and adolescents, according to the British Hypertension Society protocol

(2012) Gabriel Wai-Kwok Yip et al.   BLOOD PRESSURE MONITORING

Urinary Plasmin Activates Collecting Duct ENaC Current in Preeclampsia

(2012) Kristian B. Buhl et al.   HYPERTENSION

The Phosphorylated Sodium Chloride Cotransporter in Urinary Exosomes Is Superior to Prostasin as a Marker for Aldosteronism

(2012) Nils van der Lubbe et al.   HYPERTENSION

Prostasin-dependent activation of epithelial Na+ channels by low plasmin concentrations

(2009) Per Svenningsen et al.   AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY

Urinary prostasin in humans: relationships among prostasin, aldosterone and epithelial sodium channel activity

(2009) Aya Koda et al.   HYPERTENSION RESEARCH

Plasmin Activates Epithelial Na+Channels by Cleaving the γ Subunit

(2008) Christopher J. Passero et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Plasmin in Nephrotic Urine Activates the Epithelial Sodium Channel

(2008) P. Svenningsen et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY