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Title
RIF1 Counteracts BRCA1-mediated End Resection during DNA Repair
Authors
Keywords
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Journal
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 288, Issue 16, Pages 11135-11143
Publisher
American Society for Biochemistry & Molecular Biology (ASBMB)
Online
2013-03-14
DOI
10.1074/jbc.m113.457440
References
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Note: Only part of the references are listed.- RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection
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- A Cell Cycle-Dependent Regulatory Circuit Composed of 53BP1-RIF1 and BRCA1-CtIP Controls DNA Repair Pathway Choice
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- Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replication
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- 53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility
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- Human exonuclease 1 and BLM helicase interact to resect DNA and initiate DNA repair
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