Cysteine Proteinase fromStreptococcus pyogenesEnables Evasion of Innate Immunity via Degradation of Complement Factors

Therapeutic options for patients with angioedema due to C1-inhibitor deficiencies: from pathophysiology to the clinic

(2012) Roberto Castelli et al.   IMMUNOPHARMACOLOGY AND IMMUNOTOXICOLOGY

Gram-positive bacterial superantigen outside-in signaling causes toxic shock syndrome

(2011) Amanda J. Brosnahan et al.   FEBS Journal

Biofilm formation or internalization into epithelial cells enable Streptococcus pyogenes to evade antibiotic eradication in patients with pharyngitis

(2011) Taiji Ogawa et al.   MICROBIAL PATHOGENESIS

Insights intoStreptococcus pyogenespathogenesis from transcriptome studies

(2010) Tomas Fiedler et al.   Future Microbiology

Gram-positive toxic shock syndromes

(2009) Emma Lappin et al.   LANCET INFECTIOUS DISEASES

SpeB of Streptococcus pyogenes Differentially Modulates Antibacterial and Receptor Activating Properties of Human Chemokines

(2009) Arne Egesten et al.   PLoS One

Complement activation and inhibition: a delicate balance

(2009) A.P. Sjöberg et al.   TRENDS IN IMMUNOLOGY

PfbA, a Novel Plasmin- and Fibronectin-binding Protein ofStreptococcus pneumoniae, Contributes to Fibronectin-dependent Adhesion and Antiphagocytosis

(2008) Masaya Yamaguchi et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Molecular and biological characterization of histidine triad protein in group A streptococci

(2008) Eiji Kunitomo et al.   MICROBES AND INFECTION

Fba, a novel fibronectin-binding protein from Streptococcus pyogenes, promotes bacterial entry into epithelial cells, and the fba gene is positively transcribed under the Mga regulator

(2003) Yutaka Terao et al.   MOLECULAR MICROBIOLOGY