Mice withTak1Deficiency in Neural Crest Lineage Exhibit Cleft Palate Associated with Abnormal Tongue Development
Published 2013 View Full Article
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Title
Mice withTak1Deficiency in Neural Crest Lineage Exhibit Cleft Palate Associated with Abnormal Tongue Development
Authors
Keywords
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Journal
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 288, Issue 15, Pages 10440-10450
Publisher
American Society for Biochemistry & Molecular Biology (ASBMB)
Online
2013-03-05
DOI
10.1074/jbc.m112.432286
References
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Related references
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- The mechanism of TGF-β signaling during palate development
- (2011) J Iwata et al. ORAL DISEASES
- Sprouty2 controls proliferation of palate mesenchymal cells via fibroblast growth factor signaling
- (2010) Kaori Matsumura et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- TGF-β mediated FGF10 signaling in cranial neural crest cells controls development of myogenic progenitor cells through tissue–tissue interactions during tongue morphogenesis
- (2010) Ryoichi Hosokawa et al. DEVELOPMENTAL BIOLOGY
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- Analysis of a gain-of-function FGFR2 Crouzon mutation provides evidence of loss of function activity in the etiology of cleft palate
- (2010) A. K. Snyder-Warwick et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Hand2 is required in the epithelium for palatogenesis in mice
- (2009) Wei Xiong et al. DEVELOPMENTAL BIOLOGY
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- (2009) Bryan C. Bjork et al. HUMAN MOLECULAR GENETICS
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- (2009) Jun-ichi Iwata et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Highly conserved non-coding elements on either side of SOX9 associated with Pierre Robin sequence
- (2009) Sabina Benko et al. NATURE GENETICS
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- (2008) Xun Xu et al. DEVELOPMENTAL CELL
- Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
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