The C5 Convertase Is Not Required for Activation of the Terminal Complement Pathway in Murine Experimental Cerebral Malaria

Digestive vacuole of Plasmodium falciparum released during erythrocyte rupture dually activates complement and coagulation

(2012) P. Dasari et al.   BLOOD

Cerebral malaria pathogenesis: revisiting parasite and host contributions

(2012) Georges Emile Raymond Grau et al.   Future Microbiology

Pathology of sickle cell disease

(2012) Janet I. Malowany et al.   SEMINARS IN DIAGNOSTIC PATHOLOGY

Cutting Edge: The Membrane Attack Complex of Complement Is Required for the Development of Murine Experimental Cerebral Malaria

(2011) T. N. Ramos et al.   JOURNAL OF IMMUNOLOGY

Complement deficiency states and associated infections

(2011) Lillemor Skattum et al.   MOLECULAR IMMUNOLOGY

Complement driven innate immune response to malaria: fuelling severe malarial diseases

(2010) Karlee L. Silver et al.   CELLULAR MICROBIOLOGY

Molecular Intercommunication between the Complement and Coagulation Systems

(2010) U. Amara et al.   JOURNAL OF IMMUNOLOGY

Genetic susceptibility to systemic lupus erythematosus protects against cerebral malaria in mice

(2010) M. Waisberg et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

γδ T cells in EAE: Early trafficking events and cytokine requirements

(2009) Jillian E. Wohler et al.   EUROPEAN JOURNAL OF IMMUNOLOGY

Dysregulation of coagulation in cerebral malaria

(2009) Christopher Alan Moxon et al.   MOLECULAR AND BIOCHEMICAL PARASITOLOGY

Deletion of both the C3a and C5a receptors fails to protect against experimental autoimmune encephalomyelitis

(2009) Theresa N. Ramos et al.   NEUROSCIENCE LETTERS

C5 deficiency and C5a or C5aR blockade protects against cerebral malaria

(2008) Samir N. Patel et al.   JOURNAL OF EXPERIMENTAL MEDICINE