Deficiency of H3K79 Histone Methyltransferase Dot1-like Protein (DOT1L) Inhibits Cell Proliferation
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Title
Deficiency of H3K79 Histone Methyltransferase Dot1-like Protein (DOT1L) Inhibits Cell Proliferation
Authors
Keywords
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Journal
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 287, Issue 8, Pages 5588-5599
Publisher
American Society for Biochemistry & Molecular Biology (ASBMB)
Online
2011-12-22
DOI
10.1074/jbc.m111.328138
References
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Related references
Note: Only part of the references are listed.- DOT1L, the H3K79 methyltransferase, is required for MLL-AF9-mediated leukemogenesis
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- (2010) Kyle M Miller et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
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- (2009) Chun Mei Wang et al. BIOGERONTOLOGY
- Multiple histone modifications in euchromatin promote heterochromatin formation by redundant mechanisms in Saccharomyces cerevisiae
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- Histone H2BK123 monoubiquitination is the critical determinant for H3K4 and H3K79 trimethylation by COMPASS and Dot1
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- Linking Cell Cycle to Histone Modifications: SBF and H2B Monoubiquitination Machinery and Cell-Cycle Regulation of H3K79 Dimethylation
- (2009) Julia M. Schulze et al. MOLECULAR CELL
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- (2009) Weiwei Dang et al. NATURE
- Impaired recruitment of the histone methyltransferase DOT1L contributes to the incomplete reactivation of tumor suppressor genes upon DNA demethylation
- (2009) F V Jacinto et al. ONCOGENE
- Ndy1/KDM2B immortalizes mouse embryonic fibroblasts by repressing theInk4a/Arflocus
- (2009) Alexandros Tzatsos et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- ES Cell Cycle Progression and Differentiation Require the Action of the Histone Methyltransferase Dot1L
- (2009) Evan R. Barry et al. STEM CELLS
- H3 K79 dimethylation marks developmental activation of the -globin gene but is reduced upon LCR-mediated high-level transcription
- (2008) T. Sawado et al. BLOOD
- H3K79 Methylation Profiles Define Murine and Human MLL-AF4 Leukemias
- (2008) Andrei V. Krivtsov et al. CANCER CELL
- Identification of p18INK4c as a Tumor Suppressor Gene in Glioblastoma Multiforme
- (2008) D. A. Solomon et al. CANCER RESEARCH
- DOT1L/KMT4 Recruitment and H3K79 Methylation Are Ubiquitously Coupled with Gene Transcription in Mammalian Cells
- (2008) D. J. Steger et al. MOLECULAR AND CELLULAR BIOLOGY
- Epigenetics in Cancer
- (2008) Manel Esteller NEW ENGLAND JOURNAL OF MEDICINE
- The Histone H3K79 Methyltransferase Dot1L Is Essential for Mammalian Development and Heterochromatin Structure
- (2008) Brendan Jones et al. PLoS Genetics
- Changes in H3K79 Methylation During Preimplantation Development in Mice
- (2007) Masatoshi Ooga et al. BIOLOGY OF REPRODUCTION
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