Bardet-Biedl Syndrome-associated Small GTPase ARL6 (BBS3) Functions at or near the Ciliary Gate and Modulates Wnt Signaling
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Title
Bardet-Biedl Syndrome-associated Small GTPase ARL6 (BBS3) Functions at or near the Ciliary Gate and Modulates Wnt Signaling
Authors
Keywords
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Journal
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 285, Issue 21, Pages 16218-16230
Publisher
American Society for Biochemistry & Molecular Biology (ASBMB)
Online
2010-03-06
DOI
10.1074/jbc.m109.070953
References
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Note: Only part of the references are listed.- Biochemical characterization of missense mutations in the Arf/Arl-family small GTPase Arl6 causing Bardet–Biedl syndrome
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- TheChlamydomonas reinhardtiiBBSome is an IFT cargo required for export of specific signaling proteins from flagella
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- Functional interactions between the ciliopathy-associated Meckel syndrome 1 (MKS1) protein and two novel MKS1-related (MKSR) proteins
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- Mechanistic insights into Bardet-Biedl syndrome, a model ciliopathy
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- elipsa is an early determinant of ciliogenesis that links the IFT particle to membrane-associated small GTPase Rab8
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- The retinitis pigmentosa 2 gene product is a GTPase-activating protein for Arf-like 3
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- Inhibition of neural crest migration underlies craniofacial dysmorphology and Hirschsprung's disease in Bardet-Biedl syndrome
- (2008) J. L. Tobin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Loss of Bardet-Biedl syndrome proteins alters the morphology and function of motile cilia in airway epithelia
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- An Essential Role for DYF-11/MIP-T3 in Assembling Functional Intraflagellar Transport Complexes
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