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miR-17-92 Cluster Targets Phosphatase and Tensin Homology and Ikaros Family Zinc Finger 4 to Promote TH17-mediated Inflammation

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Cessation of CCL2 inhibition accelerates breast cancer metastasis by promoting angiogenesis

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Origins of Metastatic Traits

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Src Family Kinases as Novel Therapeutic Targets to Treat Breast Cancer Brain Metastases

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A Secreted PTEN Phosphatase That Enters Cells to Alter Signaling and Survival

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Melanoma exosomes educate bone marrow progenitor cells toward a pro-metastatic phenotype through MET

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PI3K Pathway Mutations and PTEN Levels in Primary and Metastatic Breast Cancer

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CCL2 recruits inflammatory monocytes to facilitate breast-tumour metastasis

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Combating trastuzumab resistance by targeting SRC, a common node downstream of multiple resistance pathways

(2011) Siyuan Zhang et al.   NATURE MEDICINE

Cross-species hybridization of microarrays for studying tumor transcriptome of brain metastasis

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Unidirectional transfer of microRNA-loaded exosomes from T cells to antigen-presenting cells

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mir-17-92, a cluster of miRNAs in the midst of the cancer network

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Cancer: Genomics of metastasis

(2010) Joe Gray   NATURE

14-3-3ζ Cooperates with ErbB2 to Promote Ductal Carcinoma In Situ Progression to Invasive Breast Cancer by Inducing Epithelial-Mesenchymal Transition

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Characterization of exosome-like vesicles released from human tracheobronchial ciliated epithelium: a possible role in innate defense

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miR-19 is a key oncogenic component of mir-17-92

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Targeted Deletion Reveals Essential and Overlapping Functions of the miR-17∼92 Family of miRNA Clusters

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Effect of Lapatinib on the Outgrowth of Metastatic Breast Cancer Cells to the Brain

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