Inactivation of GSK3β and activation of NF-κB pathway via Axl represents an important mediator of tumorigenesis in esophageal squamous cell carcinoma
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Title
Inactivation of GSK3β and activation of NF-κB pathway via Axl represents an important mediator of tumorigenesis in esophageal squamous cell carcinoma
Authors
Keywords
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Journal
MOLECULAR BIOLOGY OF THE CELL
Volume 26, Issue 5, Pages 821-831
Publisher
American Society for Cell Biology (ASCB)
Online
2015-01-08
DOI
10.1091/mbc.e14-04-0868
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Note: Only part of the references are listed.- Mechanisms of AXL overexpression and function in Imatinib-resistant chronic myeloid leukemia cells
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