(−)-Epigallocatechin-3-gallate blocks nicotine-induced matrix metalloproteinase-9 expression and invasiveness via suppression of NF-κB and AP-1 in endothelial cells

Nicotine- and Tar-Free Cigarette Smoke Induces Cell Damage Through Reactive Oxygen Species Newly Generated by PKC-Dependent Activation of NADPH Oxidase

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ERK-associated changes of AP-1 proteins during fear extinction

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Epigenetic Effects and Molecular Mechanisms of Tumorigenesis Induced by Cigarette Smoke: An Overview

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Epigallocatechin-3-gallate (EGCG) downregulates gelatinase-B (MMP-9) by involvement of FAK/ERK/NFκB and AP-1 in the human breast cancer cell line MDA-MB-231

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Roles of matrix metalloproteinases in cancer progression and their pharmacological targeting

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ROS-NFκΒ mediates TGF-β1-induced expression of urokinase-type plasminogen activator, matrix metalloproteinase-9 and cell invasion

(2010) Nicolas Tobar et al.   MOLECULAR AND CELLULAR BIOCHEMISTRY

Transcription Factor Egr-1 Is Essential for Maximal Matrix Metalloproteinase-9 Transcription by Tumor Necrosis Factor  

(2010) S. Y. Shin et al.   MOLECULAR CANCER RESEARCH

Evidence for the involvement of matrix metalloproteinases in the cardiovascular effects produced by nicotine

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Nicotine Promotes Tumor Growth and Metastasis in Mouse Models of Lung Cancer

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Matrix metalloproteinase inhibitors as anticancer agents

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