RORα, a Potential Tumor Suppressor and Therapeutic Target of Breast Cancer
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Title
RORα, a Potential Tumor Suppressor and Therapeutic Target of Breast Cancer
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 13, Issue 12, Pages 15755-15766
Publisher
MDPI AG
Online
2012-11-27
DOI
10.3390/ijms131215755
References
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Note: Only part of the references are listed.- ROR Suppresses Breast Tumor Invasion by Inducing SEMA3F Expression
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- Transcription factor RORα is critical for nuocyte development
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- Regulation of p53 Stability and Apoptosis by a ROR Agonist
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- Activation of the Canonical Wnt/β-Catenin Pathway in ATF3-Induced Mammary Tumors
- (2011) Leqin Yan et al. PLoS One
- Identification of SR1078, a Synthetic Agonist for the Orphan Nuclear Receptors RORα and RORγ
- (2010) Yongjun Wang et al. ACS Chemical Biology
- Development of a Lung Cancer Therapeutic Based on the Tumor Suppressor MicroRNA-34
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- Characterization of the Core Mammalian Clock Component, NPAS2, as a REV-ERBα/RORα Target Gene
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- RORα Attenuates Wnt/β-Catenin Signaling by PKCα-Dependent Phosphorylation in Colon Cancer
- (2010) Ji Min Lee et al. MOLECULAR CELL
- Melatonin inhibits mitogenic cross-talk between retinoic acid-related orphan receptor alpha (RORα) and ERα in MCF-7 human breast cancer cells
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- Activation of Aromatase Expression by Retinoic Acid Receptor-related Orphan Receptor (ROR) α in Breast Cancer Cells
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- The nuclear receptor RORα exerts a bi-directional regulation of IL-6 in resting and reactive astrocytes
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- Role of melatonin in the epigenetic regulation of breast cancer
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- ABL2/ARG Tyrosine Kinase Mediates SEMA3F-induced RhoA Inactivation and Cytoskeleton Collapse in Human Glioma Cells
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