Suppression of AKT expression by miR-153 produced anti-tumor activity in lung cancer

miR-153 Supports Colorectal Cancer Progression via Pleiotropic Effects That Enhance Invasion and Chemotherapeutic Resistance

(2013) L. Zhang et al.   CANCER RESEARCH

MicroRNAs: key players of taxane resistance and their therapeutic potential in human cancers

(2013) Shi-Yun Cui et al.   JOURNAL OF CELLULAR AND MOLECULAR MEDICINE

MicroRNA-153 is tumor suppressive in glioblastoma stem cells

(2013) Shiguang Zhao et al.   MOLECULAR BIOLOGY REPORTS

Downregulation of miR-153 contributes to epithelial-mesenchymal transition and tumor metastasis in human epithelial cancer

(2012) Qin Xu et al.   CARCINOGENESIS

miR-337-3p and Its Targets STAT3 and RAP1A Modulate Taxane Sensitivity in Non-Small Cell Lung Cancers

(2012) Liqin Du et al.   PLoS One

Upregulation of miR-153 promotes cell proliferation via downregulation of the PTEN tumor suppressor gene in human prostate cancer

(2012) Ziqing Wu et al.   PROSTATE

MiR-34a chemosensitizes bladder cancer cells to cisplatin treatment regardless of p53-Rb pathway status

(2011) Ruth L. Vinall et al.   INTERNATIONAL JOURNAL OF CANCER

miR-153 sensitized the K562 cells to As2O3-induced apoptosis

(2011) Li Liu et al.   MEDICAL ONCOLOGY

MicroRNA-451 functions as a tumor suppressor in human non-small cell lung cancer by targeting ras-related protein 14 (RAB14)

(2011) R Wang et al.   ONCOGENE

Modulation of K-Ras-Dependent Lung Tumorigenesis by MicroRNA-21

(2010) Mark E. Hatley et al.   CANCER CELL

Cancer Statistics, 2009

(2009) A. Jemal et al.   CA-A CANCER JOURNAL FOR CLINICIANS

miR-221&222 Regulate TRAIL Resistance and Enhance Tumorigenicity through PTEN and TIMP3 Downregulation

(2009) Michela Garofalo et al.   CANCER CELL

Dithiolethione compounds inhibit Akt signaling in human breast and lung cancer cells by increasing PP2A activity

(2009) C H Switzer et al.   ONCOGENE

MiR-107 and MiR-185 Can Induce Cell Cycle Arrest in Human Non Small Cell Lung Cancer Cell Lines

(2009) Yukari Takahashi et al.   PLoS One