Loss of gene function as a consequence of human papillomavirus DNA integration

CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression

(2011) Z Liu et al.   CELL DEATH AND DIFFERENTIATION

CASZ1b, the Short Isoform of CASZ1 Gene, Coexpresses with CASZ1a during Neurogenesis and Suppresses Neuroblastoma Cell Growth

(2011) Zhihui Liu et al.   PLoS One

Frequent genomic structural alterations at HPV insertion sites in cervical carcinoma

(2010) Martine Peter et al.   JOURNAL OF PATHOLOGY

Identification of human papillomavirus type 16 integration sites in high-grade precancerous cervical lesions

(2009) Mihaela Matovina et al.   GYNECOLOGIC ONCOLOGY

Mechanism of Genomic Instability in Cells Infected with the High-Risk Human Papillomaviruses

(2009) Meelis Kadaja et al.   PLoS Pathogens

Characterization of Naturally Occurring HPV16 Integration Sites Isolated from Cervical Keratinocytes under Noncompetitive Conditions

(2008) K. L. Dall et al.   CANCER RESEARCH

The Majority of Viral-Cellular Fusion Transcripts in Cervical Carcinomas Cotranscribe Cellular Sequences of Known or Predicted Genes

(2008) I. Kraus et al.   CANCER RESEARCH

Vertebrate CASTOR Is Required for Differentiation of Cardiac Precursor Cells at the Ventral Midline

(2008) Kathleen S. Christine et al.   DEVELOPMENTAL CELL

Identity, origin, and migration of peripheral glial cells in the Drosophila embryo

(2008) Christian M. von Hilchen et al.   MECHANISMS OF DEVELOPMENT