Inhibitory effects of antagonists of growth hormone-releasing hormone on growth and invasiveness of PC3 human prostate cancer
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Inhibitory effects of antagonists of growth hormone-releasing hormone on growth and invasiveness of PC3 human prostate cancer
Authors
Keywords
-
Journal
INTERNATIONAL JOURNAL OF CANCER
Volume 132, Issue 4, Pages 755-765
Publisher
Wiley
Online
2012-07-09
DOI
10.1002/ijc.27716
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Antagonists of growth hormone-releasing hormone inhibit growth of androgen-independent prostate cancer through inactivation of ERK and Akt kinases
- (2012) F. G. Rick et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Inhibitory effects of antagonists of growth hormone releasing hormone on experimental prostate cancers are associated with upregulation of wild-type p53 and decrease in p21 and mutant p53 proteins
- (2011) Anton Stangelberger et al. PROSTATE
- Control of Advanced Cancer: The Road to Chronicity
- (2011) Agustin Lage et al. International Journal of Environmental Research and Public Health
- GHRH antagonists reduce the invasive and metastatic potential of human cancer cell lines in vitro
- (2010) Szabolcs Bellyei et al. CANCER LETTERS
- Nuclear localization of vasoactive intestinal peptide (VIP) receptors in human breast cancer
- (2010) Ana Valdehita et al. PEPTIDES
- p120ctn and P-Cadherin but Not E-Cadherin Regulate Cell Motility and Invasion of DU145 Prostate Cancer Cells
- (2010) Sandra Kümper et al. PLoS One
- The relationship between proangiogenic gene expression levels in prostate cancer and their prognostic value for clinical outcomes
- (2010) Ryutaro Mori et al. PROSTATE
- E-cadherin, β-catenin, and ZEB1 in malignant progression of cancer
- (2009) Otto Schmalhofer et al. CANCER AND METASTASIS REVIEWS
- Osteopontin induces β-catenin signaling through activation of Akt in prostate cancer cells
- (2009) Brian W. Robertson et al. EXPERIMENTAL CELL RESEARCH
- Vasoactive intestinal peptide behaves as a pro-metastatic factor in human prostate cancer cells
- (2009) Ana B. Fernández-Martínez et al. PROSTATE
- The assessment of angiogenesis and fibroblastic stromagenesis in hyperplastic and pre-invasive breast lesions
- (2008) Kitty Pavlakis et al. BMC CANCER
- Knocking down gene expression for growth hormone-releasing hormone inhibits proliferation of human cancer cell lines
- (2008) N Barabutis et al. BRITISH JOURNAL OF CANCER
- RhoC Promotes Metastasis via Activation of the Pyk2 Pathway in Prostate Cancer
- (2008) M. Iiizumi et al. CANCER RESEARCH
- Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype
- (2008) D. Sarrio et al. CANCER RESEARCH
- New Treatment Approaches for Prostate Cancer Based on Peptide Analogues
- (2008) Anton Stangelberger et al. EUROPEAN UROLOGY
- Essential role of p21/waf1 in the mediation of the anti-proliferative effects of GHRH antagonist JMR-132
- (2008) Aspasia-Athina Volakaki et al. JOURNAL OF MOLECULAR ENDOCRINOLOGY
- Multiple Cellular Mechanisms Related to Cyclin A1 in Prostate Cancer Invasion and Metastasis
- (2008) Barbara Wegiel et al. JNCI-Journal of the National Cancer Institute
- Antioxidant activity of growth hormone-releasing hormone antagonists in LNCaP human prostate cancer line
- (2008) N. Barabutis et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Dose-dependent growth inhibition in vivo of PC-3 prostate cancer with a reduction in tumoral growth factors after therapy with GHRH antagonist MZ-J-7-138
- (2008) Elmar Heinrich et al. PROSTATE
- Antagonists of growth-hormone-releasing hormone: an emerging new therapy for cancer
- (2007) Andrew V Schally et al. Nature clinical practice. Endocrinology & metabolism
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started