Smad2 and Smad3 are redundantly essential for the suppression of iNOS synthesis in macrophages by regulating IRF3 and STAT1 pathways

The ECS(SPSB) E3 ubiquitin ligase is the master regulator of the lifetime of inducible nitric-oxide synthase

(2011) Kazuma Matsumoto et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Suppression of SOCS3 in macrophages prevents cancer metastasis by modifying macrophage phase and MCP2/CCL8 induction

(2011) Kiyokazu Hiwatashi et al.   CANCER LETTERS

Histone 3 Lysine 9 (H3K9) Methyltransferase Recruitment to the Interleukin-2 (IL-2) Promoter Is a Mechanism of Suppression of IL-2 Transcription by the Transforming Growth Factor-β-Smad Pathway

(2011) Yu Wakabayashi et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

TLR Regulation of SPSB1 Controls Inducible Nitric Oxide Synthase Induction

(2011) R. S. Lewis et al.   JOURNAL OF IMMUNOLOGY

Smad2 and Smad3 Are Redundantly Essential for the TGF- -Mediated Regulation of Regulatory T Plasticity and Th1 Development

(2010) T. Takimoto et al.   JOURNAL OF IMMUNOLOGY

Silencing ofSOCS1in macrophages suppresses tumor development by enhancing antitumor inflammation

(2009) Masayuki Hashimoto et al.   CANCER SCIENCE

Cyclic Adenosine Monophosphate Suppresses the Transcription of Proinflammatory Cytokines via the Phosphorylated c-Fos Protein

(2009) Keiko Koga et al.   IMMUNITY

Cross-talk between IFN-α and TGF-β1signaling pathways in preneoplastic rat liver

(2008) María De Luján Alvarez* et al.   GROWTH FACTORS

TRAF6 and MEKK1 Play a Pivotal Role in the RIG-I-like Helicase Antiviral Pathway

(2008) Ryoko Yoshida et al.   JOURNAL OF BIOLOGICAL CHEMISTRY