Pioglitazone reduces inflammation through inhibition of NF-κB in polymicrobial sepsis
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Pioglitazone reduces inflammation through inhibition of NF-κB in polymicrobial sepsis
Authors
Keywords
-
Journal
Innate Immunity
Volume 20, Issue 5, Pages 519-528
Publisher
SAGE Publications
Online
2013-09-13
DOI
10.1177/1753425913501565
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Peroxisome Proliferator–Activated Receptor-γ Activation Prevents Sepsis-Related Cardiac Dysfunction and Mortality In Mice
- (2013) Konstantinos Drosatos et al. Circulation-Heart Failure
- Effect of Eritoran, an Antagonist of MD2-TLR4, on Mortality in Patients With Severe Sepsis
- (2013) Steven M. Opal et al. JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
- Activation of Peroxisome Proliferator-Activated Receptorγby Rosiglitazone Inhibits Lipopolysaccharide-Induced Release of High Mobility Group Box 1
- (2012) Jung Seok Hwang et al. MEDIATORS OF INFLAMMATION
- PPAR gamma activation protects the brain against microvascular dysfunction in sepsis
- (2012) C.V. Araújo et al. MICROVASCULAR RESEARCH
- Short-Term High Fat Feeding Increases Organ Injury and Mortality After Polymicrobial Sepsis
- (2012) Jennifer M. Kaplan et al. Obesity
- Pioglitazone limits myocardial infarct size, activates Akt, and upregulates cPLA2 and COX-2 in a PPAR-γ-independent manner
- (2011) Yochai Birnbaum et al. BASIC RESEARCH IN CARDIOLOGY
- Adiponectin Attenuates Lipopolysaccharide-Induced Acute Lung Injury through Suppression of Endothelial Cell Activation
- (2011) J. M. Konter et al. JOURNAL OF IMMUNOLOGY
- Effects of Pioglitazone on Survival and Omental Adipocyte Function in Mice with Sepsis Induced by Cecal Ligation and Puncture
- (2011) Yukihiro Tsujimura et al. JOURNAL OF SURGICAL RESEARCH
- Decreased hepatic peroxisome proliferator-activated receptor-γ contributes to increased sensitivity to endotoxin in obstructive jaundice
- (2011) Xin Lv WORLD JOURNAL OF GASTROENTEROLOGY
- Peroxisome Proliferator-Activated Receptor δ Regulates Inflammation via NF-κB Signaling in Polymicrobial Sepsis
- (2010) Basilia Zingarelli et al. AMERICAN JOURNAL OF PATHOLOGY
- Pioglitazone Attenuates Allergic Inflammation and Induces Production of Regulatory T Lymphocytes
- (2010) Weihua Wang et al. American Journal of Rhinology & Allergy
- Original Article: Peroxisome proliferator-activated receptor and AMP-activated protein kinase agonists protect against lethal influenza virus challenge in mice
- (2010) Carson E. Moseley et al. Influenza and Other Respiratory Viruses
- Changes in peroxisome proliferator-activated receptor-gamma activity in children with septic shock
- (2009) Jennifer M. Kaplan et al. INTENSIVE CARE MEDICINE
- TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection
- (2009) J. R. Aldridge et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Adiponectin deficiency is associated with severe polymicrobial sepsis, high inflammatory cytokine levels, and high mortality
- (2009) Yoshitaka Uji et al. SURGERY
- Adiponectin deficiency promotes endothelial activation and profoundly exacerbates sepsis-related mortality
- (2008) Hwee Teoh et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Cyclooxygenase-2 induction by adiponectin is regulated by a sphingosine kinase-1 dependent mechanism in cardiac myocytes
- (2008) Yasumasa Ikeda et al. FEBS LETTERS
- Pioglitazone reduces systematic inflammation and improves mortality in apolipoprotein E knockout mice with sepsis
- (2008) Go Haraguchi et al. INTENSIVE CARE MEDICINE
- Pleiotrophic and anti-inflammatory effects of pioglitazone precede the metabolic activity in type 2 diabetic patients with coronary artery disease
- (2007) T. Forst et al. ATHEROSCLEROSIS
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started