Carbon dioxide is largely responsible for the acute inflammatory effects of tobacco smoke

Carbon dioxide inhalation causes pulmonary inflammation

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Induced TRPC1 expression increases protein phosphatase 2A sensitizing intestinal epithelial cells to apoptosis through inhibition of NF-κB activation

(2008) Bernard S. Marasa et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Hyperosmotic stress contributes to mouse colonic inflammation through the methylation of protein phosphatase 2A

(2008) Laurent Schwartz et al.   AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY

Lipopolysaccharide activates NF-κB by TLR4-Bcl10-dependent and independent pathways in colonic epithelial cells

(2008) Sumit Bhattacharyya et al.   AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY

Identification of PP2A as a crucial regulator of the NF-κB feedback loop: its inhibition by UVB turns NF-κB into a pro-apoptotic factor

(2008) S Barisic et al.   CELL DEATH AND DIFFERENTIATION

Inflammation in the development of lung cancer: epidemiological evidence

(2008) Eric A Engels   Expert Review of Anticancer Therapy

Hyperosmolarity causes inflammation through the methylation of protein phosphatase 2A

(2008) M. Abolhassani et al.   INFLAMMATION RESEARCH

Cancer-related inflammation

(2008) Alberto Mantovani et al.   NATURE

Constituents in tobacco and smoke emissions from Canadian cigarettes

(2008) D Hammond et al.   TOBACCO CONTROL