Opposing Effects of Smoking in Ulcerative Colitis and Crohnʼs Disease May Be Explained by Differential Effects on Dendritic Cells
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Title
Opposing Effects of Smoking in Ulcerative Colitis and Crohnʼs Disease May Be Explained by Differential Effects on Dendritic Cells
Authors
Keywords
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Journal
INFLAMMATORY BOWEL DISEASES
Volume 20, Issue 5, Pages 800-810
Publisher
Oxford University Press (OUP)
Online
2014-04-01
DOI
10.1097/mib.0000000000000018
References
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- (2012) Arwed Hostmann et al. IMMUNOLOGY
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- Cigarette Smoke Extract (CSE) Delays NOD2 Expression and Affects NOD2/RIPK2 Interactions in Intestinal Epithelial Cells
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- Differential development of plasmacytoid dendritic cells in Th1- and Th2-like cytokine milieus
- (2010) K. Bratke et al. ALLERGY
- Tolerogenic plasmacytoid DC
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- Cigarette smoke modulates rhinovirus-induced airway epithelial cell chemokine production
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- Chronic nicotine stimulation modulates the immune response of mucosal T cells to Th1-dominant pattern via nAChR by upregulation of Th1-specific transcriptional factor
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