Interleukin-1 Receptor Signaling Is Required To Overcome the Effects of Pertussis Toxin and for Efficient Infection- or Vaccination-Induced Immunity againstBordetella pertussis

Inflammasome Activation by Adenylate Cyclase Toxin Directs Th17 Responses and Protection against Bordetella pertussis

(2010) A. Dunne et al.   JOURNAL OF IMMUNOLOGY

Monocyte-Derived Interleukin-10 Depresses the Bordetella pertussis- Specific Gamma Interferon Response in Vaccinated Infants

(2009) V. Dirix et al.   Clinical and Vaccine Immunology

The O Antigen Is a Critical Antigen for the Development of a Protective Immune Response to Bordetella parapertussis

(2009) X. Zhang et al.   INFECTION AND IMMUNITY

Cellular effectors mediating Th17-dependent clearance of pneumococcal colonization in mice

(2009) Zhe Zhang et al.   JOURNAL OF CLINICAL INVESTIGATION

Genetically Detoxified Pertussis Toxin Induces Th1/Th17 Immune Response through MAPKs and IL-10-Dependent Mechanisms

(2009) M. Nasso et al.   JOURNAL OF IMMUNOLOGY

O Antigen Allows B. parapertussis to Evade B. pertussis Vaccine–Induced Immunity by Blocking Binding and Functions of Cross-Reactive Antibodies

(2009) Xuqing Zhang et al.   PLoS One

Pertussis Toxin Stimulates IL-17 Production in Response to Bordetella pertussis Infection in Mice

(2009) Charlotte Andreasen et al.   PLoS One

Interleukin-1 Receptor Signaling Protects Mice from Lethal Intestinal Damage Caused by the Attaching and Effacing Pathogen Citrobacter rodentium

(2008) S. L. Lebeis et al.   INFECTION AND IMMUNITY

Pertussis Toxin Inhibits Early Chemokine Production To Delay Neutrophil Recruitment in Response to Bordetella pertussis Respiratory Tract Infection in Mice

(2008) C. Andreasen et al.   INFECTION AND IMMUNITY