Aryl hydrocarbon receptor signaling regulates NF-κB RelB activation during dendritic-cell differentiation
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Title
Aryl hydrocarbon receptor signaling regulates NF-κB RelB activation during dendritic-cell differentiation
Authors
Keywords
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Journal
IMMUNOLOGY AND CELL BIOLOGY
Volume 91, Issue 9, Pages 568-575
Publisher
Wiley
Online
2013-09-03
DOI
10.1038/icb.2013.43
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Note: Only part of the references are listed.- Aryl hydrocarbon receptor contributes to the MEK/ERK-dependent maintenance of the immature state of human dendritic cells
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- (2012) R. Sibilano et al. JOURNAL OF IMMUNOLOGY
- In Situ Vaccination with CD204 Gene-Silenced Dendritic Cell, not Unmodified Dendritic Cell, Enhances Radiation Therapy of Prostate Cancer
- (2012) C. Guo et al. MOLECULAR CANCER THERAPEUTICS
- Impact of culture medium on maturation of bone marrow-derived murine dendritic cells via the aryl hydrocarbon receptor
- (2012) Anne Ilchmann et al. MOLECULAR IMMUNOLOGY
- You AhR what you eat?
- (2012) B Paige Lawrence et al. NATURE IMMUNOLOGY
- Control of RelB during dendritic cell activation integrates canonical and noncanonical NF-κB pathways
- (2012) Vincent F-S Shih et al. NATURE IMMUNOLOGY
- SU5416, a VEGF Receptor Inhibitor and Ligand of the AHR, Represents a New Alternative for Immunomodulation
- (2012) Joshua D. Mezrich et al. PLoS One
- Aryl Hydrocarbon Receptor-Mediated Perturbations in Gene Expression during Early Stages of CD4+ T-cell Differentiation
- (2012) Diana Rohlman et al. Frontiers in Immunology
- Immunotherapy with Costimulatory Dendritic Cells To Control Autoimmune Inflammation
- (2011) Brendan J. O’Sullivan et al. JOURNAL OF IMMUNOLOGY
- Opposing Biological Functions of Tryptophan Catabolizing Enzymes During Intracellular Infection
- (2011) Senad Divanovic et al. JOURNAL OF INFECTIOUS DISEASES
- AHR drives the development of gut ILC22 cells and postnatal lymphoid tissues via pathways dependent on and independent of Notch
- (2011) Jacob S Lee et al. NATURE IMMUNOLOGY
- In Vivo Dioxin Favors Interleukin-22 Production by Human CD4+ T Cells in an Aryl Hydrocarbon Receptor (AhR)-Dependent Manner
- (2011) Nicolò Costantino Brembilla et al. PLoS One
- Natural Aryl Hydrocarbon Receptor Ligands Control Organogenesis of Intestinal Lymphoid Follicles
- (2011) E. A. Kiss et al. SCIENCE
- Dioxin and immune regulation
- (2010) Nikki B. Marshall et al. Annals of the New York Academy of Sciences
- γδ T cells protect against lung fibrosis via IL-22
- (2010) Philip L. Simonian et al. JOURNAL OF EXPERIMENTAL MEDICINE
- The aryl hydrocarbon receptor interacts with c-Maf to promote the differentiation of type 1 regulatory T cells induced by IL-27
- (2010) Lionel Apetoh et al. NATURE IMMUNOLOGY
- Aryl hydrocarbon receptor negatively regulates dendritic cell immunogenicity via a kynurenine-dependent mechanism
- (2010) N. T. Nguyen et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Aryl Hydrocarbon Receptor Activation Reduces Dendritic Cell Function during Influenza Virus Infection
- (2010) Guang-Bi Jin et al. TOXICOLOGICAL SCIENCES
- Functional and phenotypic effects of AhR activation in inflammatory dendritic cells
- (2010) Jaishree Bankoti et al. TOXICOLOGY AND APPLIED PHARMACOLOGY
- Scavenger receptors: role in innate immunity and microbial pathogenesis
- (2009) Thomas Areschoug et al. CELLULAR MICROBIOLOGY
- Aryl Hydrocarbon Receptor Activation Inhibits In Vitro Differentiation of Human Monocytes and Langerhans Dendritic Cells
- (2009) B. Platzer et al. JOURNAL OF IMMUNOLOGY
- Langerhans Cell Maturation and Contact Hypersensitivity Are Impaired in Aryl Hydrocarbon Receptor-Null Mice
- (2009) B. Jux et al. JOURNAL OF IMMUNOLOGY
- Ah receptor represses acute-phase response gene expression without binding to its cognate response element
- (2009) Rushang D Patel et al. LABORATORY INVESTIGATION
- Hypersensitivity of Aryl Hydrocarbon Receptor-Deficient Mice to Lipopolysaccharide-Induced Septic Shock
- (2009) H. Sekine et al. MOLECULAR AND CELLULAR BIOLOGY
- Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from TH-17, TH1 and TH2 cells
- (2009) Sara Trifari et al. NATURE IMMUNOLOGY
- Aryl hydrocarbon receptor signaling mediates expression of indoleamine 2,3-dioxygenase
- (2008) Christoph F.A. Vogel et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Ah receptor and NF-κB interplay on the stage of epigenome
- (2008) Yanan Tian BIOCHEMICAL PHARMACOLOGY
- A new cross-talk between the aryl hydrocarbon receptor and RelB, a member of the NF-κB family
- (2008) Christoph F.A. Vogel et al. BIOCHEMICAL PHARMACOLOGY
- Cyclic AMP plays a critical role in C3a-receptor-mediated regulation of dendritic cells in antigen uptake and T-cell stimulation
- (2008) K. Li et al. BLOOD
- The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins
- (2008) Marc Veldhoen et al. NATURE
- Control of Treg and TH17 cell differentiation by the aryl hydrocarbon receptor
- (2008) Francisco J. Quintana et al. NATURE
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