Journal
IMMUNITY
Volume 33, Issue 4, Pages 578-580Publisher
CELL PRESS
DOI: 10.1016/j.immuni.2010.10.003
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- NIAID NIH HHS [R00 AI072955] Funding Source: Medline
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The ability of Toll-like receptors (TLRs) to activate innate immunity depends on their transport to pathogen-containing organelles In this issue of Immunity, Husebye et al (2010) report that delivery of TLR4 to phagosomes occurs via a recycling endosome intermediate, which is controlled by the GTPase Rab11a
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