Endothelial cells from humans and mice with polycystic kidney disease are characterized by polyploidy and chromosome segregation defects through survivin down-regulation

Polyploidy impairs human aortic endothelial cell function and is prevented by nicotinamide phosphoribosyltransferase

(2009) Nica M. Borradaile et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Aurora B-Mediated Abscission Checkpoint Protects against Tetraploidization

(2009) Patrick Steigemann et al.   CELL

Ciliary Polycystin-2 Is a Mechanosensitive Calcium Channel Involved in Nitric Oxide Signaling Cascades

(2009) Wissam A. AbouAlaiwi et al.   CIRCULATION RESEARCH

Requirement of Bardet-Biedl syndrome proteins for leptin receptor signaling

(2009) Seongjin Seo et al.   HUMAN MOLECULAR GENETICS

A mechanism linking extra centrosomes to chromosomal instability

(2009) Neil J. Ganem et al.   NATURE

INPP5E mutations cause primary cilium signaling defects, ciliary instability and ciliopathies in human and mouse

(2009) Monique Jacoby et al.   NATURE GENETICS

Adult Type 3 Adenylyl Cyclase–Deficient Mice Are Obese

(2009) Zhenshan Wang et al.   PLoS One

Polycystic Kidney Disease

(2008) Peter C. Harris et al.   Annual Review of Medicine

Centrosome overduplication and mitotic instability in PKD2 transgenic lines

(2008) S BURTEY et al.   CELL BIOLOGY INTERNATIONAL

Deciphering the Endothelial Shear Stress Sensor

(2008) Robert E. Poelmann et al.   CIRCULATION

Endothelial Cilia Are Fluid Shear Sensors That Regulate Calcium Signaling and Nitric Oxide Production Through Polycystin-1

(2008) Surya M. Nauli et al.   CIRCULATION

Loss of polycystin-1 causes centrosome amplification and genomic instability

(2008) Lorenzo Battini et al.   HUMAN MOLECULAR GENETICS

Deletion of IFT20 in the mouse kidney causes misorientation of the mitotic spindle and cystic kidney disease

(2008) Julie A. Jonassen et al.   JOURNAL OF CELL BIOLOGY

Endothelial primary cilia in areas of disturbed flow are at the base of atherosclerosis

(2007) Kim Van der Heiden et al.   ATHEROSCLEROSIS