Suppression of Ca2+ signaling in a mouse model of Best disease

Missense Mutations in a Retinal Pigment Epithelium Protein, Bestrophin-1, Cause Retinitis Pigmentosa

(2009) Alice E. Davidson et al.   AMERICAN JOURNAL OF HUMAN GENETICS

Best1 Is a Gene Regulated by Nerve Injury and Required for Ca2+-Activated Cl- Current Expression in Axotomized Sensory Neurons

(2009) M. Boudes et al.   JOURNAL OF NEUROSCIENCE

Calcium-activated chloride currents in olfactory sensory neurons from mice lacking bestrophin-2

(2009) Simone Pifferi et al.   JOURNAL OF PHYSIOLOGY-LONDON

Functional roles of bestrophins in ocular epithelia

(2009) Alan D. Marmorstein et al.   PROGRESS IN RETINAL AND EYE RESEARCH

Biallelic Mutation of BEST1 Causes a Distinct Retinopathy in Humans

(2008) Rosemary Burgess et al.   AMERICAN JOURNAL OF HUMAN GENETICS

Expression, localization, and functional properties of Bestrophin 3 channel isolated from mouse heart

(2008) Kate E. O'Driscoll et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

The Best Disease-Linked Cl- Channel hBest1 Regulates CaV1 (L-type) Ca2+ Channels via src-Homology-Binding Domains

(2008) K. Yu et al.   JOURNAL OF NEUROSCIENCE

Molecular Physiology of Bestrophins: Multifunctional Membrane Proteins Linked to Best Disease and Other Retinopathies

(2008) H. Criss Hartzell et al.   PHYSIOLOGICAL REVIEWS