Cardiomyocyte specific adipose triglyceride lipase overexpression prevents doxorubicin induced cardiac dysfunction in female mice

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Doxorubicin treatment inhibits PPARγ and may induce lipotoxicity by mimicking a type 2 diabetes-like condition in rodent models

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Perilipin 5, a Lipid Droplet-binding Protein, Protects Heart from Oxidative Burden by Sequestering Fatty Acid from Excessive Oxidation

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Myocardial triacylglycerol metabolism

(2012) Petra C. Kienesberger et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Effects of doxorubicin on myocardial expression of apolipoprotein-B

(2012) Bjorn Redfors et al.   SCANDINAVIAN CARDIOVASCULAR JOURNAL

Development of doxorubicin-induced chronic cardiotoxicity in the B6C3F1 mouse model

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Myocardial ATGL Overexpression Decreases the Reliance on Fatty Acid Oxidation and Protects against Pressure Overload-Induced Cardiac Dysfunction

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Potential Biomarkers in Mouse Myocardium of Doxorubicin-Induced Cardiomyopathy: A Metabonomic Method and Its Application

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Cardiotoxicity of anthracycline agents for the treatment of cancer: Systematic review and meta-analysis of randomised controlled trials

(2010) Lesley A Smith et al.   BMC CANCER

Preferential Oxidation of Triacylglyceride-Derived Fatty Acids in Heart Is Augmented by the Nuclear Receptor PPARα

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Efficacy and toxicity of nonpegylated liposomal doxorubicin in breast cancer

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