标题
Structural basis of drugs that increase cardiac inward rectifier Kir2.1 currents
作者
关键词
-
出版物
CARDIOVASCULAR RESEARCH
Volume 104, Issue 2, Pages 337-346
出版商
Oxford University Press (OUP)
发表日期
2014-09-10
DOI
10.1093/cvr/cvu203
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Propafenone blocks human cardiac Kir2.x channels by decreasing the negative electrostatic charge in the cytoplasmic pore
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- Chronic atrial fibrillation up-regulates β1-Adrenoceptors affecting repolarizing currents and action potential duration
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- Inhibition of Cardiac Ca 2+ Release Channels (RyR2) Determines Efficacy of Class I Antiarrhythmic Drugs in Catecholaminergic Polymorphic Ventricular Tachycardia
- (2011) Hyun Seok Hwang et al. Circulation-Arrhythmia and Electrophysiology
- Flecainide Therapy Reduces Exercise-Induced Ventricular Arrhythmias in Patients With Catecholaminergic Polymorphic Ventricular Tachycardia
- (2011) Christian van der Werf et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- The anti-protozoal drug pentamidine blocks KIR2.x-mediated inward rectifier current by entering the cytoplasmic pore region of the channel
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- (2010) Martin Tristani-Firouzi et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Flecainide increases Kir2.1 currents by interacting with cysteine 311, decreasing the polyamine-induced rectification
- (2010) R. Caballero et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Nitric Oxide Increases Cardiac I K1 by Nitrosylation of Cysteine 76 of Kir2.1 Channels
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- Cardiac strong inward rectifier potassium channels
- (2009) Justus M.B. Anumonwo et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Inhibition of IK,ACh current may contribute to clinical efficacy of class I and class III antiarrhythmic drugs in patients with atrial fibrillation
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- Reduction of complex ventricular ectopy and improvement in exercise capacity with flecainide therapy in Andersen-Tawil syndrome
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