Ligustilide Ameliorates Memory Deficiency in APP/PS1 Transgenic Mice via Restoring Mitochondrial Dysfunction
出版年份 2018 全文链接
标题
Ligustilide Ameliorates Memory Deficiency in APP/PS1 Transgenic Mice via Restoring Mitochondrial Dysfunction
作者
关键词
-
出版物
Biomed Research International
Volume 2018, Issue -, Pages 1-15
出版商
Hindawi Limited
发表日期
2018-07-11
DOI
10.1155/2018/4606752
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Decreased Proteolytic Activity of the Mitochondrial Amyloid-β Degrading Enzyme, PreP Peptidasome, in Alzheimer's Disease Brain Mitochondria
- (2018) Nyosha Alikhani et al. JOURNAL OF ALZHEIMERS DISEASE
- Tetrahydrohyperforin Decreases Cholinergic Markers associated with Amyloid-β Plaques, 4-Hydroxynonenal Formation, and Caspase-3 Activation in AβPP/PS1 Mice
- (2018) Francisco J. Carvajal et al. JOURNAL OF ALZHEIMERS DISEASE
- Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer’s disease
- (2017) Ramesh Kandimalla et al. HUMAN MOLECULAR GENETICS
- Mitochondrial FOXO3a is involved in amyloid β peptide-induced mitochondrial dysfunction
- (2016) Chun Shi et al. JOURNAL OF BIOENERGETICS AND BIOMEMBRANES
- Protection against β-amyloid-induced neurotoxicity by naturally occurring Z-ligustilide through the concurrent regulation of p38 and PI3-K/Akt pathways
- (2016) Wei Xu et al. NEUROCHEMISTRY INTERNATIONAL
- Sodium Tanshinone IIA Sulfonate Attenuates Scopolamine-Induced Cognitive Dysfunctions via Improving Cholinergic System
- (2016) Qing-Qing Xu et al. Biomed Research International
- Increased neuronal PreP activity reduces Aβ accumulation, attenuates neuroinflammation and improves mitochondrial and synaptic function in Alzheimer disease's mouse model
- (2015) Du Fang et al. HUMAN MOLECULAR GENETICS
- OPA1-dependent cristae modulation is essential for cellular adaptation to metabolic demand
- (2014) D. A. Patten et al. EMBO JOURNAL
- Balance and Stability of Synaptic Structures during Synaptic Plasticity
- (2014) Daniel Meyer et al. NEURON
- Changes in the brain and plasma Aβ peptide levels with age and its relationship with cognitive impairment in the APPswe/PS1dE9 mouse model of Alzheimer’s disease
- (2014) M. Izco et al. NEUROSCIENCE
- Molecular Basis of Substrate Recognition and Degradation by Human Presequence Protease
- (2014) John V. King et al. STRUCTURE
- Longitudinal behavioral changes in the APP/PS1 transgenic Alzheimer's Disease model
- (2013) Sherry A. Ferguson et al. BEHAVIOURAL BRAIN RESEARCH
- Key Role of Mitochondria in Alzheimer’s Disease Synaptic Dysfunction
- (2013) Virve Cavallucci et al. CURRENT PHARMACEUTICAL DESIGN
- Klotho upregulation contributes to the neuroprotection of ligustilide in an Alzheimer's disease mouse model
- (2013) Xi Kuang et al. NEUROBIOLOGY OF AGING
- Cyclophilin D Deficiency Rescues Axonal Mitochondrial Transport in Alzheimer’s Neurons
- (2013) Lan Guo et al. PLoS One
- Porcine synapsin 1: SYN1 gene analysis and functional characterization of the promoter
- (2013) Claus Hedegaard et al. FEBS Open Bio
- Cell Surface Expression of the Major Amyloid-β Peptide (Aβ)-degrading Enzyme, Neprilysin, Depends on Phosphorylation by Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase Kinase (MEK) and Dephosphorylation by Protein Phosphatase 1a
- (2012) Naomasa Kakiya et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Fusion and Fission: Interlinked Processes Critical for Mitochondrial Health
- (2011) David C. Chan Annual Review of Genetics
- Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: Implications to mitochondria-targeted antioxidant therapeutics
- (2011) P. Hemachandra Reddy et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Z-ligustilide isolated from Radix Angelicae sinensis ameliorates the memory impairment induced by scopolamine in mice
- (2011) Li-Ling Cheng et al. FITOTERAPIA
- The role of mitochondria in neurodegenerative diseases
- (2011) Massimiliano Filosto et al. JOURNAL OF NEUROLOGY
- Cilostazol prevents amyloid β peptide25-35-induced memory impairment and oxidative stress in mice
- (2010) Masayuki Hiramatsu et al. BRITISH JOURNAL OF PHARMACOLOGY
- Statins Promote the Degradation of Extracellular Amyloid β-Peptide by Microglia via Stimulation of Exosome-associated Insulin-degrading Enzyme (IDE) Secretion
- (2010) Irfan Y. Tamboli et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Alzheimer's Disease
- (2010) Henry W. Querfurth et al. NEW ENGLAND JOURNAL OF MEDICINE
- Amelioration of cognitive deficits and neurodegeneration by curcumin in rat model of sporadic dementia of Alzheimer's type (SDAT)☆
- (2009) Tauheed Ishrat et al. EUROPEAN NEUROPSYCHOPHARMACOLOGY
- Mitochondria and Alzheimer’s disease: amyloid-β peptide uptake and degradation by the presequence protease, hPreP
- (2009) Nyosha Alikhani et al. JOURNAL OF BIOENERGETICS AND BIOMEMBRANES
- Protective effects of [Gly14]-Humanin on β-amyloid-induced PC12 cell death by preventing mitochondrial dysfunction
- (2009) Hui Jin et al. NEUROCHEMISTRY INTERNATIONAL
- Protective effect of Z-ligustilide against amyloid β-induced neurotoxicity is associated with decreased pro-inflammatory markers in rat brains
- (2009) Xi Kuang et al. PHARMACOLOGY BIOCHEMISTRY AND BEHAVIOR
- Amyloid- overproduction causes abnormal mitochondrial dynamics via differential modulation of mitochondrial fission/fusion proteins
- (2008) X. Wang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The amyloid -peptide is imported into mitochondria via the TOM import machinery and localized to mitochondrial cristae
- (2008) C. A. Hansson Petersen et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- ADAM-10 over-expression increases cortical synaptogenesis
- (2006) Karen F.S. Bell et al. NEUROBIOLOGY OF AGING
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started