Effect of biomechanical stress on endogenous antioxidant networks in bovine articular cartilage
出版年份 2017 全文链接
标题
Effect of biomechanical stress on endogenous antioxidant networks in bovine articular cartilage
作者
关键词
-
出版物
JOURNAL OF ORTHOPAEDIC RESEARCH
Volume -, Issue -, Pages -
出版商
Wiley
发表日期
2017-09-11
DOI
10.1002/jor.23728
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Hydrogen peroxide as a central redox signaling molecule in physiological oxidative stress: Oxidative eustress
- (2017) Helmut Sies Redox Biology
- Combining Targeted Metabolomic Data with a Model of Glucose Metabolism: Toward Progress in Chondrocyte Mechanotransduction
- (2017) Daniel Salinas et al. PLoS One
- Oxidative Stress Promotes Peroxiredoxin Hyperoxidation and Attenuates Pro-survival Signaling in Aging Chondrocytes
- (2016) John A. Collins et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Ageing and the pathogenesis of osteoarthritis
- (2016) Richard F. Loeser et al. Nature Reviews Rheumatology
- Injurious Loading of Articular Cartilage Compromises Chondrocyte Respiratory Function
- (2016) Mitchell C. Coleman et al. Arthritis & Rheumatology
- Glutathione S-transferase pi modulates NF-κB activation and pro-inflammatory responses in lung epithelial cells
- (2016) Jane T. Jones et al. Redox Biology
- Mechanical overloading causes mitochondrial superoxide and SOD2 imbalance in chondrocytes resulting in cartilage degeneration
- (2015) Masato Koike et al. Scientific Reports
- TRPV4 as a therapeutic target for joint diseases
- (2014) Amy L. McNulty et al. NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY
- The Mechanobiology of Articular Cartilage: Bearing the Burden of Osteoarthritis
- (2014) Johannah Sanchez-Adams et al. Current Rheumatology Reports
- Using exomarkers to assess mitochondrial reactive species in vivo
- (2013) Angela Logan et al. BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS
- Strain-dependent oxidant release in articular cartilage originates from mitochondria
- (2013) M. J. Brouillette et al. Biomechanics and Modeling in Mechanobiology
- Applied osmotic loading for promoting development of engineered cartilage
- (2013) Sonal R. Sampat et al. JOURNAL OF BIOMECHANICS
- Moderate dynamic compression inhibits pro-catabolic response of cartilage to mechanical injury, tumor necrosis factor-α and interleukin-6, but accentuates degradation above a strain threshold
- (2013) Y. Li et al. OSTEOARTHRITIS AND CARTILAGE
- TRPV4 channel activation improves the tensile properties of self-assembled articular cartilage constructs
- (2012) Sriram V. Eleswarapu et al. Acta Biomaterialia
- High Dietary Fat Selectively Increases Catalase Expression within Cardiac Mitochondria
- (2012) Paul M. Rindler et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Mechanical stress and ATP synthesis are coupled by mitochondrial oxidants in articular cartilage
- (2012) Katherine J. Wolff et al. JOURNAL OF ORTHOPAEDIC RESEARCH
- Mitochondrial electron transport and glycolysis are coupled in articular cartilage
- (2012) J.A. Martin et al. OSTEOARTHRITIS AND CARTILAGE
- X-ROS Signaling: Rapid Mechano-Chemo Transduction in Heart
- (2011) B. L. Prosser et al. SCIENCE
- Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease
- (2010) J. L. Scott et al. ANNALS OF THE RHEUMATIC DISEASES
- Crosstalk of reactive oxygen species and NF-κB signaling
- (2010) Michael J Morgan et al. CELL RESEARCH
- Oxidant conditioning protects cartilage from mechanically induced damage
- (2010) Prem Ramakrishnan et al. JOURNAL OF ORTHOPAEDIC RESEARCH
- Cyclic Stretch, Reactive Oxygen Species, and Vascular Remodeling
- (2009) Konstantin G. Birukov ANTIOXIDANTS & REDOX SIGNALING
- Mechanical load inhibits IL-1 induced matrix degradation in articular cartilage
- (2009) P.A. Torzilli et al. OSTEOARTHRITIS AND CARTILAGE
- Biomechanical Thresholds Regulate Inflammation through the NF-κB Pathway: Experiments and Modeling
- (2009) Jin Nam et al. PLoS One
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