标题
Lamin A/C-dependent interaction with 53BP1 promotes cellular responses to DNA damage
作者
关键词
-
出版物
AGING CELL
Volume 14, Issue 2, Pages 162-169
出版商
Wiley
发表日期
2015-01-24
DOI
10.1111/acel.12258
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection
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- A Cell Cycle-Dependent Regulatory Circuit Composed of 53BP1-RIF1 and BRCA1-CtIP Controls DNA Repair Pathway Choice
- (2013) Cristina Escribano-Díaz et al. MOLECULAR CELL
- 53BP1 is a reader of the DNA-damage-induced H2A Lys 15 ubiquitin mark
- (2013) Amélie Fradet-Turcotte et al. NATURE
- 53BP1 Regulates DSB Repair Using Rif1 to Control 5' End Resection
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- Rif1 Prevents Resection of DNA Breaks and Promotes Immunoglobulin Class Switching
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- B-type lamins and their elusive roles in metazoan cell proliferation and senescence
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- A promiscuous biotin ligase fusion protein identifies proximal and interacting proteins in mammalian cells
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- Playing the End Game: DNA Double-Strand Break Repair Pathway Choice
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- Conserved cysteine residues in the mammalian lamin A tail are essential for cellular responses to ROS generation
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- A dual role for A-type lamins in DNA double-strand break repair
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- A new pathway that regulates 53BP1 stability implicates Cathepsin L and vitamin D in DNA repair
- (2011) Ignacio Gonzalez-Suarez et al. EMBO JOURNAL
- Deciphering arginine methylation: Tudor tells the tale
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- 53BP1 Inhibits Homologous Recombination in Brca1-Deficient Cells by Blocking Resection of DNA Breaks
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- Assembly and function of DNA double-strand break repair foci in mammalian cells
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- 53BP1 regulates DNA resection and the choice between classical and alternative end joining during class switch recombination
- (2010) Anne Bothmer et al. JOURNAL OF EXPERIMENTAL MEDICINE
- The DNA Damage Response: Making It Safe to Play with Knives
- (2010) Alberto Ciccia et al. MOLECULAR CELL
- 53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair
- (2010) Angela T. Noon et al. NATURE CELL BIOLOGY
- Cellular senescence: unravelling complexity
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- Novel roles for A-type lamins in telomere biology and the DNA damage response pathway
- (2009) Ignacio Gonzalez-Suarez et al. EMBO JOURNAL
- The DNA-damage response in human biology and disease
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- 53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility
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- Activation of ATM depends on chromatin interactions occurring before induction of DNA damage
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