标题
Hypoxia and tissue destruction in pulmonary TB
作者
关键词
-
出版物
THORAX
Volume 71, Issue 12, Pages 1145-1153
出版商
BMJ
发表日期
2016-06-01
DOI
10.1136/thoraxjnl-2015-207402
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Mycobacterium tuberculosisdysregulates MMP/TIMP balance to drive rapid cavitation and unrestrained bacterial proliferation
- (2014) André Kübler et al. JOURNAL OF PATHOLOGY
- Hypoxia Inducible Factor Signaling Modulates Susceptibility to Mycobacterial Infection via a Nitric Oxide Dependent Mechanism
- (2013) Philip M. Elks et al. PLoS Pathogens
- Doxycycline and HIV Infection Suppress Tuberculosis-induced Matrix Metalloproteinases
- (2012) Naomi F. Walker et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Prolyl Hydroxylase 3 (PHD3) Modulates Catabolic Effects of Tumor Necrosis Factor-α (TNF-α) on Cells of the Nucleus Pulposus through Co-activation of Nuclear Factor κB (NF-κB)/p65 Signaling
- (2012) Nobuyuki Fujita et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- PET of Hypoxia: Current and Future Perspectives
- (2012) S. Carlin et al. JOURNAL OF NUCLEAR MEDICINE
- Common patterns and disease-related signatures in tuberculosis and sarcoidosis
- (2012) J. Maertzdorf et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Single-subject statistical mapping of acute brain hypoxia in the rat following middle cerebral artery occlusion: A microPET study
- (2011) Masashi Takasawa et al. EXPERIMENTAL NEUROLOGY
- MMP-1 drives immunopathology in human tuberculosis and transgenic mice
- (2011) Paul Elkington et al. JOURNAL OF CLINICAL INVESTIGATION
- Mouse Model of Necrotic Tuberculosis Granulomas Develops Hypoxic Lesions
- (2011) Jamie Harper et al. JOURNAL OF INFECTIOUS DISEASES
- Mycobacterium tuberculosis-infected human monocytes down-regulate microglial MMP-2 secretion in CNS tuberculosis via TNFα, NFκB, p38 and caspase 8 dependent pathways
- (2011) Justin A Green et al. Journal of Neuroinflammation
- Hypoxia and Inflammation
- (2011) Holger K. Eltzschig et al. NEW ENGLAND JOURNAL OF MEDICINE
- Hypoxia differentially affects IL-1 -stimulated MMP-1 and MMP-13 expression of fibroblast-like synoviocytes in an HIF-1 -dependent manner
- (2011) Y.-A. Lee et al. RHEUMATOLOGY
- Identifying hypoxia in human tumors: A correlation study between18F-FMISO PET and the Eppendorf oxygen-sensitive electrode
- (2010) Lise Saksø Mortensen et al. ACTA ONCOLOGICA
- Caseation of human tuberculosis granulomas correlates with elevated host lipid metabolism
- (2010) Mi-Jeong Kim et al. EMBO Molecular Medicine
- Hypoxia Induces an Immunodominant Target of Tuberculosis Specific T Cells Absent from Common BCG Vaccines
- (2010) Hannah Priyadarshini Gideon et al. PLoS Pathogens
- Significance of nitroimidazole compounds and hypoxia-inducible factor-1 for imaging tumor hypoxia
- (2009) Shinae Kizaka-Kondoh et al. CANCER SCIENCE
- Unique Roles of DosT and DosS in DosR Regulon Induction and Mycobacterium tuberculosis Dormancy
- (2009) R. W. Honaker et al. INFECTION AND IMMUNITY
- Pharmacokinetic Analysis of Hypoxia 18F-Fluoromisonidazole Dynamic PET in Head and Neck Cancer
- (2009) W. Wang et al. JOURNAL OF NUCLEAR MEDICINE
- Tuberculous Granuloma Induction via Interaction of a Bacterial Secreted Protein with Host Epithelium
- (2009) H. E. Volkman et al. SCIENCE
- Tuberculous Granulomas Are Hypoxic in Guinea Pigs, Rabbits, and Nonhuman Primates
- (2008) L. E. Via et al. INFECTION AND IMMUNITY
- NF-κB links innate immunity to the hypoxic response through transcriptional regulation of HIF-1α
- (2008) Jordi Rius et al. NATURE
- Oxygen-Independent Stabilization of Hypoxia Inducible Factor (HIF)-1 during RSV Infection
- (2008) Helene A. Haeberle et al. PLoS One
- The Influence of Host and Bacterial Genotype on the Development of Disseminated Disease with Mycobacterium tuberculosis
- (2008) Maxine Caws et al. PLoS Pathogens
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