Inhibition of Interleukin-21 prolongs the survival through the promotion of wound healing after myocardial infarction
出版年份 2021 全文链接
标题
Inhibition of Interleukin-21 prolongs the survival through the promotion of wound healing after myocardial infarction
作者
关键词
IL-21, Myocardial infarction, Wound healing, Myocardial inflammation
出版物
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
Volume 159, Issue -, Pages 48-61
出版商
Elsevier BV
发表日期
2021-06-16
DOI
10.1016/j.yjmcc.2021.06.006
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- IL-21 promotes allergic airway inflammation by driving apoptosis of FoxP3+ regulatory T cells
- (2019) Luigi Tortola et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- IL-21/type I interferon interplay regulates neutrophil-dependent innate immune responses to Staphylococcus aureus
- (2019) Rosanne Spolski et al. eLife
- Matrix metalloproteinase-12 produced by Ly6Clow macrophages prolongs the survival after myocardial infarction by preventing neutrophil influx
- (2019) Akihiko Kubota et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Heart failure as interstitial cancer: emergence of a malignant fibroblast phenotype
- (2019) Kelsie E. Oatmen et al. Nature Reviews Cardiology
- Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality
- (2018) Merry L Lindsey et al. CARDIOVASCULAR RESEARCH
- The extracellular matrix in myocardial injury, repair, and remodeling
- (2017) Nikolaos G. Frangogiannis JOURNAL OF CLINICAL INVESTIGATION
- Pathologically expanded peripheral T helper cell subset drives B cells in rheumatoid arthritis
- (2017) Deepak A. Rao et al. NATURE
- Macrophage function in tissue repair and remodeling requires IL-4 or IL-13 with apoptotic cells
- (2017) Lidia Bosurgi et al. SCIENCE
- Deletion of CD28 Co-stimulatory Signals Exacerbates Left Ventricular Remodeling and Increases Cardiac Rupture After Myocardial Infarction
- (2016) Akihiko Kubota et al. CIRCULATION JOURNAL
- Defining the Cardiac Fibroblast
- (2016) Malina J. Ivey et al. CIRCULATION JOURNAL
- The Biological Basis for Cardiac Repair After Myocardial Infarction
- (2016) Sumanth D. Prabhu et al. CIRCULATION RESEARCH
- Cardiac myofibroblast engulfment of dead cells facilitates recovery after myocardial infarction
- (2016) Michio Nakaya et al. JOURNAL OF CLINICAL INVESTIGATION
- Inflammation as a Driver of Adverse Left Ventricular Remodeling After Acute Myocardial Infarction
- (2016) Peter C. Westman et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Loss of Interleukin-21 Receptor Activation in Hypoxic Endothelial Cells Impairs Perfusion Recovery After Hindlimb Ischemia
- (2015) T. Wang et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- BLT1 antagonist LSN2792613 reduces infarct size in a mouse model of myocardial ischaemia–reperfusion injury
- (2015) Vince C. de Hoog et al. CARDIOVASCULAR RESEARCH
- Revisiting Cardiac Cellular CompositionNovelty and Significance
- (2015) Alexander R. Pinto et al. CIRCULATION RESEARCH
- Intratumoral Delivery of IL-21 Overcomes Anti-Her2/Neu Resistance through Shifting Tumor-Associated Macrophages from M2 to M1 Phenotype
- (2015) Meng Xu et al. JOURNAL OF IMMUNOLOGY
- Ly-6C high Monocytes Depend on Nr4a1 to Balance Both Inflammatory and Reparative Phases in the Infarcted Myocardium
- (2014) Ingo Hilgendorf et al. CIRCULATION RESEARCH
- Differential Contribution of Monocytes to Heart Macrophages in Steady-State and After Myocardial Infarction
- (2014) Timo Heidt et al. CIRCULATION RESEARCH
- Foxp3 + CD4 + T Cells Improve Healing After Myocardial Infarction by Modulating Monocyte/Macrophage Differentiation
- (2014) Johannes Weirather et al. CIRCULATION RESEARCH
- Sox5 and c-Maf cooperatively induce Th17 cell differentiation via RORγt induction as downstream targets of Stat3
- (2014) Shigeru Tanaka et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Interleukin-21: a double-edged sword with therapeutic potential
- (2014) Rosanne Spolski et al. NATURE REVIEWS DRUG DISCOVERY
- Enhanced Efferocytosis of Apoptotic Cardiomyocytes Through Myeloid-Epithelial-Reproductive Tyrosine Kinase Links Acute Inflammation Resolution to Cardiac Repair After Infarction
- (2013) Elaine Wan et al. CIRCULATION RESEARCH
- The Cytokines IL-21 and GM-CSF Have Opposing Regulatory Roles in the Apoptosis of Conventional Dendritic Cells
- (2013) Chi-Keung Wan et al. IMMUNITY
- Executive Summary: Heart Disease and Stroke Statistics—2012 Update
- (2012) et al. CIRCULATION
- Activation of CD4 + T Lymphocytes Improves Wound Healing and Survival After Experimental Myocardial Infarction in Mice
- (2012) Ulrich Hofmann et al. CIRCULATION
- Regulatory Role of Dendritic Cells in Postinfarction Healing and Left Ventricular Remodeling
- (2012) Atsushi Anzai et al. CIRCULATION
- Interleukin-21 – A biomarker of importance in predicting myocardial function following acute infarction?
- (2012) Robin A.P. Weir et al. CYTOKINE
- Interleukin-21 Is Critically Required in Autoimmune and Allogeneic Responses to Islet Tissue in Murine Models
- (2011) Helen M. McGuire et al. DIABETES
- A Subset of Interleukin-21+ Chemokine Receptor CCR9+ T Helper Cells Target Accessory Organs of the Digestive System in Autoimmunity
- (2011) Helen M. McGuire et al. IMMUNITY
- Monocytes: Protagonists of Infarct Inflammation and Repair After Myocardial Infarction
- (2010) Matthias Nahrendorf et al. CIRCULATION
- A Positive Feedback Loop of IL-21 Signaling Provoked by Homeostatic CD4+CD25- T Cell Expansion Is Essential for the Development of Arthritis in Autoimmune K/BxN Mice
- (2009) E. Jang et al. JOURNAL OF IMMUNOLOGY
- c-Maf activates the promoter and enhancer of the IL-21 gene, and TGF-β inhibits c-Maf-induced IL-21 production in CD4+ T cells
- (2009) Yukiko Hiramatsu et al. JOURNAL OF LEUKOCYTE BIOLOGY
- Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites
- (2009) F. K. Swirski et al. SCIENCE
- Development and characterization of IL-21–producing CD4+T cells
- (2008) Akira Suto et al. JOURNAL OF EXPERIMENTAL MEDICINE
Become a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get StartedAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started