Herpes simplex virus encoded ICP6 protein forms functional amyloid assemblies with necroptosis-associated host proteins
出版年份 2020 全文链接
标题
Herpes simplex virus encoded ICP6 protein forms functional amyloid assemblies with necroptosis-associated host proteins
作者
关键词
ICP6, RIP homotypic interaction motif, Amyloid, Necroptosis
出版物
BIOPHYSICAL CHEMISTRY
Volume 269, Issue -, Pages 106524
出版商
Elsevier BV
发表日期
2020-12-15
DOI
10.1016/j.bpc.2020.106524
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Identification of MLKL membrane translocation as a checkpoint in necroptotic cell death using Monobodies
- (2020) Emma J. Petrie et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Cryo-EM structure of an amyloid fibril formed by full-length human prion protein
- (2020) Li-Qiang Wang et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
- NSs amyloid formation is associated with the virulence of Rift Valley fever virus in mice
- (2020) Psylvia Léger et al. Nature Communications
- Varicella zoster virus encodes a viral decoy RHIM to inhibit cell death
- (2020) Megan Steain et al. PLoS Pathogens
- The role of necroptosis in cancer biology and therapy
- (2019) Yitao Gong et al. Molecular Cancer
- The Killer Pseudokinase Mixed Lineage Kinase Domain-Like Protein (MLKL)
- (2019) James M. Murphy Cold Spring Harbor Perspectives in Biology
- Single-molecule detection on a portable 3D-printed microscope
- (2019) James W. P. Brown et al. Nature Communications
- Cysteine oxidation triggers amyloid fibril formation of the tumor suppressor p16INK4A
- (2019) Christoph Göbl et al. Redox Biology
- Herpesviruses induce aggregation and selective autophagy of host signalling proteins NEMO and RIPK1 as an immune-evasion mechanism
- (2019) Elena Muscolino et al. Nature Microbiology
- The Structure of the Necrosome RIPK1-RIPK3 Core, a Human Hetero-Amyloid Signaling Complex
- (2018) Miguel Mompeán et al. CELL
- Necroptosis: a regulated inflammatory mode of cell death
- (2018) Yogesh K. Dhuriya et al. Journal of Neuroinflammation
- DNA-induced liquid phase condensation of cGAS activates innate immune signaling
- (2018) Mingjian Du et al. SCIENCE
- Species-independent contribution of ZBP1/DAI/DLM-1-triggered necroptosis in host defense against HSV1
- (2018) Hongyan Guo et al. Cell Death & Disease
- Viral M45 and necroptosis‐associated proteins form heteromeric amyloid assemblies
- (2018) Chi LL Pham et al. EMBO REPORTS
- The structure of a β2-microglobulin fibril suggests a molecular basis for its amyloid polymorphism
- (2018) Matthew G. Iadanza et al. Nature Communications
- Peptidoglycan-Sensing Receptors Trigger the Formation of Functional Amyloids of the Adaptor Protein Imd to Initiate Drosophila NF-κB Signaling
- (2017) Anni Kleino et al. IMMUNITY
- Apoptosis and Necroptosis as Host Defense Strategies to Prevent Viral Infection
- (2017) Megan H. Orzalli et al. TRENDS IN CELL BIOLOGY
- The Structure and Dynamics of Higher-Order Assemblies: Amyloids, Signalosomes, and Granules
- (2016) Hao Wu et al. CELL
- Identification of a novel cell death-inducing domain reveals that fungal amyloid-controlled programmed cell death is related to necroptosis
- (2016) Asen Daskalov et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Herpes Simplex Virus Suppresses Necroptosis in Human Cells
- (2015) Hongyan Guo et al. Cell Host & Microbe
- RIP1/RIP3 Binding to HSV-1 ICP6 Initiates Necroptosis to Restrict Virus Propagation in Mice
- (2015) Zhe Huang et al. Cell Host & Microbe
- Necroptosis: The Trojan horse in cell autonomous antiviral host defense
- (2015) Edward S. Mocarski et al. VIROLOGY
- The Putative Herpes Simplex Virus 1 Chaperone Protein UL32 Modulates Disulfide Bond Formation during Infection
- (2014) Brandon S. Albright et al. JOURNAL OF VIROLOGY
- Direct activation of RIP3/MLKL-dependent necrosis by herpes simplex virus 1 (HSV-1) protein ICP6 triggers host antiviral defense
- (2014) X. Wang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Necroptosis in Immunity and Ischemia-Reperfusion Injury
- (2013) A. Linkermann et al. AMERICAN JOURNAL OF TRANSPLANTATION
- GROMACS 4.5: a high-throughput and highly parallel open source molecular simulation toolkit
- (2013) Sander Pronk et al. BIOINFORMATICS
- The RIP1/RIP3 Necrosome Forms a Functional Amyloid Signaling Complex Required for Programmed Necrosis
- (2012) Jixi Li et al. CELL
- DAI/ZBP1/DLM-1 Complexes with RIP3 to Mediate Virus-Induced Programmed Necrosis that Is Targeted by Murine Cytomegalovirus vIRA
- (2012) Jason W. Upton et al. Cell Host & Microbe
- HSV Infection Induces Production of ROS, which Potentiate Signaling from Pattern Recognition Receptors: Role for S-glutathionylation of TRAF3 and 6
- (2011) Regina Gonzalez-Dosal et al. PLoS Pathogens
- Intracellular Redox Compartments: Mechanisms and Significances
- (2010) Thomas Kietzmann ANTIOXIDANTS & REDOX SIGNALING
- Herpes simplex virus induces neural oxidative damage via microglial cell Toll-like receptor-2
- (2010) Scott J Schachtele et al. Journal of Neuroinflammation
- Improved side-chain torsion potentials for the Amber ff99SB protein force field
- (2010) Kresten Lindorff-Larsen et al. PROTEINS-STRUCTURE FUNCTION AND BIOINFORMATICS
- The Role of the Kinases RIP1 and RIP3 in TNF-Induced Necrosis
- (2010) P. Vandenabeele et al. Science Signaling
- DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-κB
- (2009) Manuele Rebsamen et al. EMBO REPORTS
- Cytomegalovirus M45 Cell Death Suppression Requires Receptor-interacting Protein (RIP) Homotypic Interaction Motif (RHIM)-dependent Interaction with RIP1
- (2008) Jason W. Upton et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Structural Analysis Reveals an Amyloid Form of the Human Papillomavirus Type 16 E1 E4 Protein and Provides a Molecular Basis for Its Accumulation
- (2008) P. B. McIntosh et al. JOURNAL OF VIROLOGY
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