Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling
出版年份 2016 全文链接
标题
Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling
作者
关键词
Myocardium, Ischemia, Reperfusion, Apoptosis, Caspases
出版物
BASIC RESEARCH IN CARDIOLOGY
Volume 111, Issue 2, Pages -
出版商
Springer Nature
发表日期
2016-02-16
DOI
10.1007/s00395-016-0537-6
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Regulatory role of CARD3 in left ventricular remodelling and dysfunction after myocardial infarction
- (2015) Liangpeng Li et al. BASIC RESEARCH IN CARDIOLOGY
- Gadd45γregulates cardiomyocyte death and post-myocardial infarction left ventricular remodelling
- (2015) Alexandre Lucas et al. CARDIOVASCULAR RESEARCH
- Cyclophilin D and myocardial ischemia–reperfusion injury: A fresh perspective
- (2015) Muhammad Rizwan Alam et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Executioner Caspase-3 and 7 Deficiency Reduces Myocyte Number in the Developing Mouse Heart
- (2015) Maria Cardona et al. PLoS One
- Apoptotic Caspases Prevent the Induction of Type I Interferons by Mitochondrial DNA
- (2014) Anthony Rongvaux et al. CELL
- Cardiomyocyte-Specific Deletion of Gsk3α Mitigates Post–Myocardial Infarction Remodeling, Contractile Dysfunction, and Heart Failure
- (2014) Firdos Ahmad et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Omentin Prevents Myocardial Ischemic Injury Through AMP-Activated Protein Kinase- and Akt-Dependent Mechanisms
- (2014) Yoshiyuki Kataoka et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- SNX13 reduction mediates heart failure through degradative sorting of apoptosis repressor with caspase recruitment domain
- (2014) Jun Li et al. Nature Communications
- Ca 2+ /Calmodulin-Dependent Protein Kinase II δ Mediates Myocardial Ischemia/Reperfusion Injury Through Nuclear Factor-κB
- (2013) Haiyun Ling et al. CIRCULATION RESEARCH
- Inhibition of Apoptosis Signal–Regulating Kinase 1 Reduces Myocardial Ischemia–Reperfusion Injury in the Mouse
- (2013) Stefano Toldo et al. Journal of the American Heart Association
- Essential Role of Caveolin-3 in Adiponectin Signalsome Formation and Adiponectin Cardioprotection
- (2012) Yajing Wang et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Mechanisms of Cell Death in Heart Disease
- (2012) Klitos Konstantinidis et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Targeting cell death in the reperfused heart: Pharmacological approaches for cardioprotection
- (2012) Martinus I.F.J. Oerlemans et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Interleukin-17A Contributes to Myocardial Ischemia/Reperfusion Injury by Regulating Cardiomyocyte Apoptosis and Neutrophil Infiltration
- (2012) Yu-Hua Liao et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Molecular and cellular mechanisms involved in cardiac remodeling after acute myocardial infarction
- (2011) Gemma Vilahur et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function
- (2011) Chris McDermott-Roe et al. NATURE
- EndoG Links Bnip3-Induced Mitochondrial Damage and Caspase-Independent DNA Fragmentation in Ischemic Cardiomyocytes
- (2011) Jisheng Zhang et al. PLoS One
- Lethal reperfusion injury in acute myocardial infarction: facts and unresolved issues
- (2009) D. Garcia-Dorado et al. CARDIOVASCULAR RESEARCH
- Prognostic Value of Myocardial Infarct Size and Contractile Reserve Using Magnetic Resonance Imaging
- (2009) Sebastian Kelle et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Constitutive COX-2 activity in cardiomyocytes confers permanent cardioprotection
- (2008) Javier Inserte et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationFind the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
Search