Do N-arachidonyl-glycine (NA-glycine) and 2-arachidonoyl glycerol (2-AG) share mode of action and the binding site on the β2subunit of GABAAreceptors?

N-arachidonoyl glycine suppresses Na+/Ca2+exchanger-mediated Ca2+entry into endothelial cells and activates BKCachannels independently of GPCRs

(2013) Alexander I Bondarenko et al.   BRITISH JOURNAL OF PHARMACOLOGY

Molecular analysis of the site for 2-arachidonylglycerol (2-AG) on the β2subunit of GABAAreceptors

(2013) Roland Baur et al.   JOURNAL OF NEUROCHEMISTRY

Structure, Function, and Modulation of GABAAReceptors

(2012) Erwin Sigel et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Molecular Sites for the Positive Allosteric Modulation of Glycine Receptors by Endocannabinoids

(2011) Gonzalo E. Yévenes et al.   PLoS One

The major central endocannabinoid directly acts at GABAA receptors

(2011) E. Sigel et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

T-Type Calcium Channel Inhibition Underlies the Analgesic Effects of the Endogenous Lipoamino Acids

(2009) G. Barbara et al.   JOURNAL OF NEUROSCIENCE

Minocycline Treatment Inhibits Microglial Activation and Alters Spinal Levels of Endocannabinoids in a Rat Model of Neuropathic Pain

(2009) Leonardo Guasti et al.   Molecular Pain

Identification of Farnesyl Pyrophosphate andN-Arachidonylglycine as Endogenous Ligands for GPR92

(2008) Da Young Oh et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

International Union of Pharmacology. LXX. Subtypes of  -Aminobutyric AcidA Receptors: Classification on the Basis of Subunit Composition, Pharmacology, and Function. Update

(2008) R. W. Olsen et al.   PHARMACOLOGICAL REVIEWS