标题
Glucose Metabolism Regulates T Cell Activation, Differentiation, and Functions
作者
关键词
-
出版物
Frontiers in Immunology
Volume 6, Issue -, Pages -
出版商
Frontiers Media SA
发表日期
2015-01-23
DOI
10.3389/fimmu.2015.00001
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Hypoxic culture conditions enhance the generation of regulatory T cells
- (2015) Thi My Anh Neildez-Nguyen et al. IMMUNOLOGY
- How Does Monocyte Metabolism Impact Inflammation and Aging During Chronic HIV Infection?
- (2014) Clovis S. Palmer et al. AIDS RESEARCH AND HUMAN RETROVIRUSES
- FADD is essential for glucose uptake and survival of thymocytes
- (2014) Xiang-Yu Zhang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- The Glucose Transporter Glut1 Is Selectively Essential for CD4 T Cell Activation and Effector Function
- (2014) Andrew N. Macintyre et al. Cell Metabolism
- Cellular Energy Metabolism in T-Lymphocytes
- (2014) Timo Gaber et al. INTERNATIONAL REVIEWS OF IMMUNOLOGY
- Metabolic Mysteries of the Inflammatory Response: T Cell Polarization and Plasticity
- (2014) Kelley M. Fracchia et al. INTERNATIONAL REVIEWS OF IMMUNOLOGY
- Metabolic Reprogramming of Macrophages
- (2014) Alex J. Freemerman et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways
- (2014) S. R. Jacobs et al. JOURNAL OF IMMUNOLOGY
- Glucose Transporter 1–Expressing Proinflammatory Monocytes Are Elevated in Combination Antiretroviral Therapy–Treated and Untreated HIV+Subjects
- (2014) Clovis S. Palmer et al. JOURNAL OF IMMUNOLOGY
- Metabolism of stromal and immune cells in health and disease
- (2014) Bart Ghesquière et al. NATURE
- Bcl-6 directly represses the gene program of the glycolysis pathway
- (2014) Kenneth J Oestreich et al. NATURE IMMUNOLOGY
- Metabolic Reprogramming towards Aerobic Glycolysis Correlates with Greater Proliferative Ability and Resistance to Metabolic Inhibition in CD8 versus CD4 T Cells
- (2014) Yilin Cao et al. PLoS One
- Monocytes as Regulators of Inflammation and HIV-Related Comorbidities during cART
- (2014) Joshua J. Anzinger et al. Journal of Immunology Research
- Increased glucose metabolic activity is associated with CD4+ T-cell activation and depletion during chronic HIV infection
- (2013) Clovis S. Palmer et al. AIDS
- Metabolic Regulation of T Lymphocytes
- (2013) Nancie J. MacIver et al. Annual Review of Immunology
- mTOR kinase inhibitors as potential cancer therapeutic drugs
- (2013) Shi-Yong Sun CANCER LETTERS
- Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis
- (2013) Chih-Hao Chang et al. CELL
- Mitochondria Are Required for Antigen-Specific T Cell Activation through Reactive Oxygen Species Signaling
- (2013) Laura A. Sena et al. IMMUNITY
- The transcription factor IRF4 is essential for TCR affinity–mediated metabolic programming and clonal expansion of T cells
- (2013) Kevin Man et al. NATURE IMMUNOLOGY
- Fueling Immunity: Insights into Metabolism and Lymphocyte Function
- (2013) E. L. Pearce et al. SCIENCE
- mTOR Signaling in Growth Control and Disease
- (2012) Mathieu Laplante et al. CELL
- mTOR, metabolism, and the regulation of T-cell differentiation and function
- (2012) Adam T. Waickman et al. IMMUNOLOGICAL REVIEWS
- Glut1-mediated glucose transport regulates HIV infection
- (2012) S. Loisel-Meyer et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Regulation of glucose metabolism in T cells: new insight into the role of Phosphoinositide 3-kinases
- (2012) David K. Finlay Frontiers in Immunology
- Control of TH17/Treg Balance by Hypoxia-Inducible Factor 1
- (2011) Eric V. Dang et al. CELL
- The many faces of Th17 cells
- (2011) Anneli Peters et al. CURRENT OPINION IN IMMUNOLOGY
- The Transcription Factor Myc Controls Metabolic Reprogramming upon T Lymphocyte Activation
- (2011) Ruoning Wang et al. IMMUNITY
- HIF1α–dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Tregcells
- (2011) Lewis Z. Shi et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Cutting Edge: Distinct Glycolytic and Lipid Oxidative Metabolic Programs Are Essential for Effector and Regulatory CD4+ T Cell Subsets
- (2011) R. D. Michalek et al. JOURNAL OF IMMUNOLOGY
- The kinase mTOR regulates the differentiation of helper T cells through the selective activation of signaling by mTORC1 and mTORC2
- (2011) Greg M Delgoffe et al. NATURE IMMUNOLOGY
- Mammalian target of rapamycin up-regulation of pyruvate kinase isoenzyme type M2 is critical for aerobic glycolysis and tumor growth
- (2011) Q. Sun et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Interleukin-7 mediates glucose utilization in lymphocytes through transcriptional regulation of the hexokinase II gene
- (2010) Mounir Chehtane et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Toll-like receptor-induced changes in glycolytic metabolism regulate dendritic cell activation
- (2010) C. M. Krawczyk et al. BLOOD
- Induction of Glucose Metabolism in Stimulated T Lymphocytes Is Regulated by Mitogen-Activated Protein Kinase Signaling
- (2010) Aimee J. Marko et al. PLoS One
- The mTOR Kinase Differentially Regulates Effector and Regulatory T Cell Lineage Commitment
- (2009) Greg M. Delgoffe et al. IMMUNITY
- mTOR signaling at a glance
- (2009) M. Laplante et al. JOURNAL OF CELL SCIENCE
- Anergic T Cells Are Metabolically Anergic
- (2009) Y. Zheng et al. JOURNAL OF IMMUNOLOGY
- mTOR regulates memory CD8 T-cell differentiation
- (2009) Koichi Araki et al. NATURE
- Normalization of obesity-associated insulin resistance through immunotherapy
- (2009) Shawn Winer et al. NATURE MEDICINE
- How Diverse--CD4 Effector T Cells and their Functions
- (2009) Y. Y. Wan et al. Journal of Molecular Cell Biology
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More