Parkinson's disease-associated genetic variation is linked to quantitative expression of inflammatory genes

Epigenetic regulation in Parkinson’s disease

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The Central Nervous System and the Gut Microbiome

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Molecular and genetic inflammation networks in major human diseases

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Parkinson-associated risk variant in distal enhancer of α-synuclein modulates target gene expression

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Integration of summary data from GWAS and eQTL studies predicts complex trait gene targets

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Using the Gene Ontology to Annotate Key Players in Parkinson’s Disease

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Enrichment of risk SNPs in regulatory regions implicate diverse tissues in Parkinson’s disease etiology

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dbSUPER: a database of super-enhancers in mouse and human genome

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PANTHER version 10: expanded protein families and functions, and analysis tools

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Motif signatures in stretch enhancers are enriched for disease-associated genetic variants

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Insights into Neuroinflammation in Parkinson’s Disease: From Biomarkers to Anti-Inflammatory Based Therapies

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Neuroinflammation in Parkinson’s disease and its potential as therapeutic target

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Mutant LRRK2 Toxicity in Neurons Depends on LRRK2 Levels and Synuclein But Not Kinase Activity or Inclusion Bodies

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The role of the LRRK2 gene in Parkinsonism

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Large-scale meta-analysis of genome-wide association data identifies six new risk loci for Parkinson's disease

(2014) Mike A Nalls et al.   NATURE GENETICS

MHCII Is Required for  -Synuclein-Induced Activation of Microglia, CD4 T Cell Proliferation, and Dopaminergic Neurodegeneration

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The Genotype-Tissue Expression (GTEx) project

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The NHGRI GWAS Catalog, a curated resource of SNP-trait associations

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Mechanisms of LRRK2-Mediated Neurodegeneration

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A pathway-based analysis provides additional support for an immune-related genetic susceptibility to Parkinson's disease

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Evaluation of Parkinson Disease Risk Variants as Expression-QTLs

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Comprehensive Research Synopsis and Systematic Meta-Analyses in Parkinson's Disease Genetics: The PDGene Database

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Imputation of sequence variants for identification of genetic risks for Parkinson's disease: a meta-analysis of genome-wide association studies

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Neuroinflammation in Parkinson's disease

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Common genetic variation in the HLA region is associated with late-onset sporadic Parkinson's disease

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Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

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Incidence and remaining lifetime risk of Parkinson disease in advanced age

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