Changes in the astrocytic aquaporin-4 and inwardly rectifying potassium channel expression in the brain of the amyotrophic lateral sclerosis SOD1G93Arat model
出版年份 2012 全文链接
标题
Changes in the astrocytic aquaporin-4 and inwardly rectifying potassium channel expression in the brain of the amyotrophic lateral sclerosis SOD1G93Arat model
作者
关键词
-
出版物
GLIA
Volume 60, Issue 12, Pages 1991-2003
出版商
Wiley
发表日期
2012-09-15
DOI
10.1002/glia.22414
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Decreased expression of the glial water channel aquaporin-4 in the intrahippocampal kainic acid model of epileptogenesis
- (2012) Darrin J. Lee et al. EXPERIMENTAL NEUROLOGY
- Potassium Dependent Regulation of Astrocyte Water Permeability Is Mediated by cAMP Signaling
- (2012) Yutong Song et al. PLoS One
- Deletion of aquaporin-4 changes the perivascular glial protein scaffold without disrupting the brain endothelial barrier
- (2011) Martine Eilert-Olsen et al. GLIA
- Laminar-specific and developmental expression of aquaporin-4 in the mouse hippocampus
- (2011) M.S. Hsu et al. NEUROSCIENCE
- Glial-conditional deletion of aquaporin-4 (Aqp4) reduces blood-brain water uptake and confers barrier function on perivascular astrocyte endfeet
- (2011) N. N. Haj-Yasein et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Monitoring systemic oxidative stress in an animal model of amyotrophic lateral sclerosis
- (2010) Francisco Javier Miana-Mena et al. JOURNAL OF NEUROLOGY
- The Role of Oxidative Stress in Amyotrophic Lateral Sclerosis and Parkinson’s Disease
- (2010) Athan Baillet et al. NEUROCHEMICAL RESEARCH
- In VivoMorphological Changes in Animal Models of Amyotrophic Lateral Sclerosis and Alzheimer's-Like Disease: MRI Approach
- (2009) Pavle R. Andjus et al. Anatomical Record-Advances in Integrative Anatomy and Evolutionary Biology
- Glutamate Transporters and the Excitotoxic Path to Motor Neuron Degeneration in Amyotrophic Lateral Sclerosis
- (2009) Emily Foran et al. ANTIOXIDANTS & REDOX SIGNALING
- Impaired blood–brain and blood–spinal cord barriers in mutant SOD1-linked ALS rat
- (2009) Charles Nicaise et al. BRAIN RESEARCH
- A systematic study of brainstem motor nuclei in a mouse model of ALS, the effects of lithium
- (2009) Michela Ferrucci et al. NEUROBIOLOGY OF DISEASE
- Altered blood–brain barrier integrity in adult aquaporin-4 knockout mice
- (2009) Jianping Zhou et al. NEUROREPORT
- AQP4 gene deletion in mice does not alter blood–brain barrier integrity or brain morphology
- (2009) S. Saadoun et al. NEUROSCIENCE
- Aquaporin-4 in brain and spinal cord oedema
- (2009) S. Saadoun et al. NEUROSCIENCE
- Amyotrophic lateral sclerosis
- (2009) Lokesh C Wijesekera et al. Orphanet Journal of Rare Diseases
- Aquaporin-4 Overexpression in Rat ALS Model
- (2008) Charles Nicaise et al. Anatomical Record-Advances in Integrative Anatomy and Evolutionary Biology
- Identification of a molecular target for glutamate regulation of astrocyte water permeability
- (2008) Eli Gunnarson et al. GLIA
- Immunohistochemical Distribution of Inwardly Rectifying K+ Channels in the Medulla Oblongata of the Rat
- (2008) Yoshio YAMAMOTO et al. JOURNAL OF VETERINARY MEDICAL SCIENCE
- ALS-causing SOD1 mutants generate vascular changes prior to motor neuron degeneration
- (2008) Zhihui Zhong et al. NATURE NEUROSCIENCE
- The Blood-Brain Barrier in Health and Chronic Neurodegenerative Disorders
- (2008) Berislav V. Zlokovic NEURON
- Aquaporin-4 independent Kir4.1 K+ channel function in brain glial cells
- (2007) Hua Zhang et al. MOLECULAR AND CELLULAR NEUROSCIENCE
- Transgenic SOD1 G93A mice develop reduced GLT-1 in spinal cord without alterations in cerebrospinal fluid glutamate levels
- (2003) Caterina Bendotti et al. JOURNAL OF NEUROCHEMISTRY
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