Oncogenes induce senescence with incomplete growth arrest and suppress the DNA damage response in immortalized cells

TGF-  signaling engages an ATM-CHK2-p53-independent RAS-induced senescence and prevents malignant transformation in human mammary epithelial cells

(2011) R. Cipriano et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Jailbreak: Oncogene-induced senescence and its evasion

(2010) Fiona K.E. MDuff et al.   CELLULAR SIGNALLING

Cellular senescence: putting the paradoxes in perspective

(2010) Judith Campisi   CURRENT OPINION IN GENETICS & DEVELOPMENT

HSP72 depletion suppresses γH2AX activation by genotoxic stresses via p53/p21 signaling

(2010) V L Gabai et al.   ONCOGENE

Defining the ATM-mediated barrier to tumorigenesis in somatic mammary cells following ErbB2 activation

(2010) Jay P. Reddy et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Genetic and Phenotypic Characteristics of Pleomorphic Lobular Carcinoma In Situ of the Breast

(2009) Yunn-Yi Chen et al.   AMERICAN JOURNAL OF SURGICAL PATHOLOGY

Molecular Distinctions between Stasis and Telomere Attrition Senescence Barriers Shown by Long-term Culture of Normal Human Mammary Epithelial Cells

(2009) J. C. Garbe et al.   CANCER RESEARCH

The ATR-Chk1 pathway plays a role in the generation of centrosome aberrations induced by Rad51C dysfunction

(2009) M. Katsura et al.   NUCLEIC ACIDS RESEARCH

Triggering Senescence Programs Suppresses Chk1 Kinase and Sensitizes Cells To Genotoxic Stresses

(2008) V. L. Gabai et al.   CANCER RESEARCH

Visualizing Spatiotemporal Dynamics of Multicellular Cell-Cycle Progression

(2008) Asako Sakaue-Sawano et al.   CELL

ATR and H2AX Cooperate in Maintaining Genome Stability under Replication Stress

(2008) Rebecca A. Chanoux et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Heat Shock Protein Hsp72 Controls Oncogene-Induced Senescence Pathways in Cancer Cells

(2008) V. L. Gabai et al.   MOLECULAR AND CELLULAR BIOLOGY

Complementary functions of ATM and H2AX in development and suppression of genomic instability

(2008) S. Zha et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA