Pseudomonas aeruginosa stimulates tyrosine phosphorylation of and TLR5 association with the MUC1 cytoplasmic tail through EGFR activation
出版年份 2015 全文链接
标题
Pseudomonas aeruginosa stimulates tyrosine phosphorylation of and TLR5 association with the MUC1 cytoplasmic tail through EGFR activation
作者
关键词
MUC1, EGFR, TLR5, <em class=EmphasisTypeItalic >Pseudomonas aeruginosa</em>, Flagellin, Normal human bronchial epithelial cells, Inflammation
出版物
INFLAMMATION RESEARCH
Volume 65, Issue 3, Pages 225-233
出版商
Springer Nature
发表日期
2015-12-09
DOI
10.1007/s00011-015-0908-8
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Suppression of Toll-like receptor-mediated innate immune responses at the ocular surface by the membrane-associated mucins MUC1 and MUC16
- (2015) B B Menon et al. Mucosal Immunology
- Targeting the Oncogenic MUC1-C Protein Inhibits Mutant EGFR-Mediated Signaling and Survival in Non-Small Cell Lung Cancer Cells
- (2014) A. Kharbanda et al. CLINICAL CANCER RESEARCH
- Activated EGFR stimulates MUC1 expression in human uterine and pancreatic cancer cell lines
- (2013) Neeraja Dharmaraj et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Activation of inflammasome signaling mediates pathology of acute P. aeruginosa pneumonia
- (2013) Taylor S. Cohen et al. JOURNAL OF CLINICAL INVESTIGATION
- Prevention of lung injury by Muc1 mucin in a mouse model of repetitive Pseudomonas aeruginosa infection
- (2012) Tsuyoshi Umehara et al. INFLAMMATION RESEARCH
- Membrane-Tethered MUC1 Mucin Is Phosphorylated by Epidermal Growth Factor Receptor in Airway Epithelial Cells and Associates with TLR5 To Inhibit Recruitment of MyD88
- (2012) K. Kato et al. JOURNAL OF IMMUNOLOGY
- MUC1 Contributes to BPDE-Induced Human Bronchial Epithelial Cell Transformation through Facilitating EGFR Activation
- (2012) Xiuling Xu et al. PLoS One
- Dissecting negative regulation of Toll-like receptor signaling
- (2012) Takeshi Kondo et al. TRENDS IN IMMUNOLOGY
- Antiinflammatory Role of MUC1 Mucin during Infection with NontypeableHaemophilus influenzae
- (2011) Yoshiyuki Kyo et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Anti-MUC1 antibody inhibits EGF receptor signaling in cancer cells
- (2011) Akinori Hisatsune et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Flagellin/TLR5 responses induce mucus hypersecretion by activating EGFR via an epithelial cell signaling cascades
- (2011) Hongmei Yu et al. EXPERIMENTAL CELL RESEARCH
- Anti-inflammatory effect of MUC1 during respiratory syncytial virus infection of lung epithelial cells in vitro
- (2010) Yusheng Li et al. AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY
- TNF-α Is a Key Regulator of MUC1, an Anti-inflammatory Molecule, during AirwayPseudomonas aeruginosaInfection
- (2010) Seongwon Choi et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- ADAM-17: the enzyme that does it all
- (2010) Monika Gooz CRITICAL REVIEWS IN BIOCHEMISTRY AND MOLECULAR BIOLOGY
- MUC1 regulates nuclear localization and function of the epidermal growth factor receptor
- (2010) B. G. Bitler et al. JOURNAL OF CELL SCIENCE
- The Identification of Markers of Macrophage Differentiation in PMA-Stimulated THP-1 Cells and Monocyte-Derived Macrophages
- (2010) Marc Daigneault et al. PLoS One
- Deletion of the Mucin-Like Molecule Muc1 Enhances Dendritic Cell Activation in Response to Toll-Like Receptor Ligands
- (2009) Marc A. Williams et al. Journal of Innate Immunity
- Pseudomonas aeruginosa LPS or Flagellin Are Sufficient to Activate TLR-Dependent Signaling in Murine Alveolar Macrophages and Airway Epithelial Cells
- (2009) Eloïse Raoust et al. PLoS One
- Increased mortality of ventilated patients with endotracheal Pseudomonas aeruginosa without clinical signs of infection*
- (2008) Hanjing Zhuo et al. CRITICAL CARE MEDICINE
- MUC1 Mucin Is a Negative Regulator of Toll-Like Receptor Signaling
- (2007) Keiko Ueno et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started