Pin1 mediates Aβ42-induced dendritic spine loss

Protein interacting with NIMA (never in mitosis A)-1 regulates axonal growth cone adhesion and spreading through myristoylated alanine-rich C kinase substrate isomerization

(2016) Lucas J. Sosa et al.   JOURNAL OF NEUROCHEMISTRY

Pin1 Modulates the Synaptic Content of NMDA Receptors via Prolyl-Isomerization of PSD-95

(2016) R. Antonelli et al.   JOURNAL OF NEUROSCIENCE

Analyzing dendritic spine pathology in Alzheimer’s disease: problems and opportunities

(2015) Mario M. Dorostkar et al.   ACTA NEUROPATHOLOGICA

Myosin Va mediates BDNF-induced postendocytic recycling of full-length TrkB and its translocation into dendritic spines

(2015) W.-H. Sui et al.   JOURNAL OF CELL SCIENCE

Pin1 cysteine-113 oxidation inhibits its catalytic activity and cellular function in Alzheimer's disease

(2015) Chun-Hau Chen et al.   NEUROBIOLOGY OF DISEASE

NGF but not BDNF overexpression protects hippocampal LTP from beta-amyloid-induced impairment

(2015) A.D. Ivanov et al.   NEUROSCIENCE

Differential calcium alterations in animal models of neurodegenerative disease: Reversal by FK506

(2015) C.R. Overk et al.   NEUROSCIENCE

Reduced Synaptic STIM2 Expression and Impaired Store-Operated Calcium Entry Cause Destabilization of Mature Spines in Mutant Presenilin Mice

(2014) Suya Sun et al.   NEURON

Isomerase Pin1 Stimulates Dephosphorylation of Tau Protein at Cyclin-dependent Kinase (Cdk5)-dependent Alzheimer Phosphorylation Sites

(2013) Taeko Kimura et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Distinct Dendritic Spine and Nuclear Phases of Calcineurin Activation after Exposure to Amyloid-  Revealed by a Novel Fluorescence Resonance Energy Transfer Assay

(2012) H.-Y. Wu et al.   JOURNAL OF NEUROSCIENCE

Cloud CPFP: A Shotgun Proteomics Data Analysis Pipeline Using Cloud and High Performance Computing

(2012) David C. Trudgian et al.   JOURNAL OF PROTEOME RESEARCH

Prolyl Isomerase Pin1 Regulates Neuronal Differentiation via  -Catenin

(2012) K. Nakamura et al.   MOLECULAR AND CELLULAR BIOLOGY

Mixed-lineage kinase 3 phosphorylates prolyl-isomerase Pin1 to regulate its nuclear translocation and cellular function

(2012) V. Rangasamy et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Death-Associated Protein Kinase 1 Phosphorylates Pin1 and Inhibits Its Prolyl Isomerase Activity and Cellular Function

(2011) Tae Ho Lee et al.   MOLECULAR CELL

Preparation of Synaptoneurosomes from Mouse Cortex using a Discontinuous Percoll-Sucrose Density Gradient

(2011) Pamela R. Westmark et al.   Jove-Journal of Visualized Experiments

CPFP: a central proteomics facilities pipeline

(2010) D. C. Trudgian et al.   BIOINFORMATICS

Amyloid   Induces the Morphological Neurodegenerative Triad of Spine Loss, Dendritic Simplification, and Neuritic Dystrophies through Calcineurin Activation

(2010) H.-Y. Wu et al.   JOURNAL OF NEUROSCIENCE

Amyloid-β oligomers impair fear conditioned memory in a calcineurin-dependent fashion in mice

(2010) Kelly T. Dineley et al.   JOURNAL OF NEUROSCIENCE RESEARCH

Calcineurin inhibition with FK506 ameliorates dendritic spine density deficits in plaque-bearing Alzheimer model mice

(2010) Anete Rozkalne et al.   NEUROBIOLOGY OF DISEASE

Pin1 and PKM  Sequentially Control Dendritic Protein Synthesis

(2010) P. R. Westmark et al.   Science Signaling

Wnt-5a/JNK Signaling Promotes the Clustering of PSD-95 in Hippocampal Neurons

(2009) Ginny G. Farías et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Alzheimer's disease: synaptic dysfunction and Aβ

(2009) Ganesh M Shankar et al.   Molecular Neurodegeneration

Soluble Oligomers of Amyloid β Protein Facilitate Hippocampal Long-Term Depression by Disrupting Neuronal Glutamate Uptake

(2009) Shaomin Li et al.   NEURON

Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

(2008) Ganesh M Shankar et al.   NATURE MEDICINE

Automated Three-Dimensional Detection and Shape Classification of Dendritic Spines from Fluorescence Microscopy Images

(2008) Alfredo Rodriguez et al.   PLoS One