Metabotropic Glutamate Receptors Protect Oligodendrocytes from Acute Ischemia in the Mouse Optic Nerve

Expression of Kir4.1 and Kir5.1 inwardly rectifying potassium channels in oligodendrocytes, the myelinating cells of the CNS

(2016) C. Brasko et al.   Brain Structure & Function

mGluR5 protect astrocytes from ischemic damage in postnatal CNS white matter

(2015) Ilaria Vanzulli et al.   CELL CALCIUM

Neurotransmitter signaling in white matter

(2014) Arthur M. Butt et al.   GLIA

White matter injury: Ischemic and nonischemic

(2014) Robert F. Fern et al.   GLIA

Astrocyte-Derived BDNF Supports Myelin Protein Synthesis after Cuprizone-Induced Demyelination

(2014) C. G. Fulmer et al.   JOURNAL OF NEUROSCIENCE

Up-regulated GLT-1 Resists Glutamate Toxicity and Attenuates Glutamate-induced Calcium Loading in Cultured Neurocytes

(2013) Chun-hua Liu et al.   BASIC & CLINICAL PHARMACOLOGY & TOXICOLOGY

TASK-1 channels in oligodendrocytes: A role in ischemia mediated disruption

(2013) Virginia Hawkins et al.   NEUROBIOLOGY OF DISEASE

Protecting White Matter From Stroke Injury

(2012) Carlos Matute et al.   STROKE

Developmental regulation of group I metabotropic glutamate receptors in the premature brain and their protective role in a rodent model of periventricular leukomalacia

(2011) Lauren L. Jantzie et al.   Neuron Glia Biology

Reduced cAMP, Akt Activation and p65-c-Rel Dimerization: Mechanisms Involved in the Protective Effects of mGluR3 Agonists in Cultured Astrocytes

(2011) Daniela Durand et al.   PLoS One

Regulated Release of BDNF by Cortical Oligodendrocytes is Mediated Through Metabotropic Glutamate Receptors and the PLC Pathway

(2009) Issa P Bagayogo et al.   ASN Neuro

Mechanisms of ATP- and glutamate-mediated calcium signaling in white matter astrocytes

(2008) Nicola Hamilton et al.   GLIA

Metabotropic glutamate receptor 5 activation inhibits microglial associated inflammation and neurotoxicity

(2008) Kimberly R. Byrnes et al.   GLIA

White Matter Vulnerability to Ischemic Injury Increases with Age Because of Enhanced Excitotoxicity

(2008) S. Baltan et al.   JOURNAL OF NEUROSCIENCE