Corticostriatal circuit defects in Hoxb8 mutant mice

Obsessive-compulsive disorder in children and adolescents: Table 1

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Pathological circuit function underlying addiction and anxiety disorders

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Microglial Dysregulation in Psychiatric Disease

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Corticostriatal connectivity and its role in disease

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Optogenetic Stimulation of Lateral Orbitofronto-Striatal Pathway Suppresses Compulsive Behaviors

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Repeated Cortico-Striatal Stimulation Generates Persistent OCD-Like Behavior

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Abnormally High Degree Connectivity of the Orbitofrontal Cortex in Obsessive-Compulsive Disorder

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Autistic-like behaviour in Scn1a +/− mice and rescue by enhanced GABA-mediated neurotransmission

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Autism-related deficits via dysregulated eIF4E-dependent translational control

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Microglia Sculpt Postnatal Neural Circuits in an Activity and Complement-Dependent Manner

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Aberrant Striatal Functional Connectivity in Children with Autism

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Translational approaches to obsessive-compulsive disorder: from animal models to clinical treatment

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Shank3 mutant mice display autistic-like behaviours and striatal dysfunction

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Heterogeneity of CNS myeloid cells and their roles in neurodegeneration

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Synaptic Pruning by Microglia Is Necessary for Normal Brain Development

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Hematopoietic Origin of Pathological Grooming in Hoxb8 Mutant Mice

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Corticostriatal functional connectivity in non-medicated patients with obsessive-compulsive disorder

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The myeloid cells of the central nervous system parenchyma

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Slitrk5 deficiency impairs corticostriatal circuitry and leads to obsessive-compulsive–like behaviors in mice

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Altered Corticostriatal Functional Connectivity in Obsessive-compulsive Disorder

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A Computational Framework for Ultrastructural Mapping of Neural Circuitry

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Habits, Rituals, and the Evaluative Brain

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TNF-alpha polymorphisms are associated with obsessive-compulsive disorder

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Obsessive–compulsive disorder and immunology: A review

(2008) Felipe Filardi da Rocha et al.   PROGRESS IN NEURO-PSYCHOPHARMACOLOGY & BIOLOGICAL PSYCHIATRY

MPTP-induced deficits in striatal synaptic plasticity are prevented by glial cell line-derived neurotrophic factor expressed via an adeno-associated viral vector

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Integrating evidence from neuroimaging and neuropsychological studies of obsessive-compulsive disorder: The orbitofronto-striatal model revisited

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