Genome-wide host-pathogen analyses reveal genetic interaction points in tuberculosis disease
出版年份 2023 全文链接
标题
Genome-wide host-pathogen analyses reveal genetic interaction points in tuberculosis disease
作者
关键词
-
出版物
Nature Communications
Volume 14, Issue 1, Pages -
出版商
Springer Science and Business Media LLC
发表日期
2023-02-02
DOI
10.1038/s41467-023-36282-w
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The influence of human genetic variation on Epstein–Barr virus sequence diversity
- (2021) Sina Rüeger et al. Scientific Reports
- Local Ancestry Adjusted Allelic Association Analysis Robustly Captures Tuberculosis Susceptibility Loci
- (2021) Yolandi Swart et al. Frontiers in Genetics
- Interaction between host genes and Mycobacterium tuberculosis lineage can affect tuberculosis severity: Evidence for coevolution?
- (2020) Michael L. McHenry et al. PLoS Genetics
- Robust barcoding and identification of Mycobacterium tuberculosis lineages for epidemiological and clinical studies
- (2020) Gary Napier et al. Genome Medicine
- Chondroitin sulfate proteoglycans as novel drivers of leucocyte infiltration in multiple sclerosis
- (2018) Erin L Stephenson et al. BRAIN
- Leveraging lung tissue transcriptome to uncover candidate causal genes in COPD genetic associations
- (2018) Maxime Lamontagne et al. HUMAN MOLECULAR GENETICS
- Genome-wide analysis of multi- and extensively drug-resistant Mycobacterium tuberculosis
- (2018) Francesc Coll et al. NATURE GENETICS
- Ecology and evolution of Mycobacterium tuberculosis
- (2018) Sebastien Gagneux NATURE REVIEWS MICROBIOLOGY
- Pathogen lineage-based genome-wide association study identified CD53 as susceptible locus in tuberculosis
- (2017) Yosuke Omae et al. JOURNAL OF HUMAN GENETICS
- Genome-to-genome analysis highlights the effect of the human innate and adaptive immune systems on the hepatitis C virus
- (2017) M Azim Ansari et al. NATURE GENETICS
- Strain-based HLA association analysis identified HLA-DRB1*09:01 associated with modern strain tuberculosis
- (2017) L. Toyo-oka et al. HLA
- Genotype Imputation with Millions of Reference Samples
- (2016) Brian L. Browning et al. AMERICAN JOURNAL OF HUMAN GENETICS
- No Significant Effect of ASAP1 Gene Variants on the Susceptibility to Tuberculosis in Chinese Population
- (2016) Xuejiao Hu et al. MEDICINE
- HLA class II sequence variants influence tuberculosis risk in populations of European ancestry
- (2016) Gardar Sveinbjornsson et al. NATURE GENETICS
- Alleles of HLA-DRB1*04 Associated with Pulmonary Tuberculosis in Amazon Brazilian Population
- (2016) Dhêmerson Souza de Lima et al. PLoS One
- Co-evolution ofMycobacterium tuberculosisandHomo sapiens
- (2015) Daniela Brites et al. IMMUNOLOGICAL REVIEWS
- A global reference for human genetic variation
- (2015) Richard A. Gibbs et al. NATURE
- Susceptibility to tuberculosis is associated with variants in the ASAP1 gene encoding a regulator of dendritic cell migration
- (2015) James Curtis et al. NATURE GENETICS
- Rapid determination of anti-tuberculosis drug resistance from whole-genome sequences
- (2015) Francesc Coll et al. Genome Medicine
- Large-scale whole genome sequencing of M. tuberculosis provides insights into transmission in a high prevalence area
- (2015) JA Guerra-Assunção et al. eLife
- A robust SNP barcode for typing Mycobacterium tuberculosis complex strains
- (2014) Francesc Coll et al. Nature Communications
- Assessment of Mycobacterium tuberculosis transmission in Oxfordshire, UK, 2007–12, with whole pathogen genome sequences: an observational study
- (2014) Timothy M Walker et al. Lancet Respiratory Medicine
- Altered IFN- -Mediated Immunity and Transcriptional Expression Patterns in N-Ethyl-N-Nitrosourea-Induced STAT4 Mutants Confer Susceptibility to Acute Typhoid-like Disease
- (2013) M. M. Eva et al. JOURNAL OF IMMUNOLOGY
- Associations Between Human Leukocyte Antigen Class I Variants and the Mycobacterium tuberculosis Subtypes Causing Disease
- (2013) Muneeb Salie et al. JOURNAL OF INFECTIOUS DISEASES
- Imputing Amino Acid Polymorphisms in Human Leukocyte Antigens
- (2013) Xiaoming Jia et al. PLoS One
- A genome wide association study of pulmonary tuberculosis susceptibility in Indonesians
- (2012) Eileen Png et al. BMC Medical Genetics
- Genome-wide association studies of tuberculosis in Asians identify distinct at-risk locus for young tuberculosis
- (2012) Surakameth Mahasirimongkol et al. JOURNAL OF HUMAN GENETICS
- Epidemiologic Consequences of Microvariation in Mycobacterium tuberculosis
- (2012) Barun Mathema et al. JOURNAL OF INFECTIOUS DISEASES
- Host–pathogen interactions revealed by human genome-wide surveys
- (2012) Chiea Chuen Khor et al. TRENDS IN GENETICS
- Variant G57E of Mannose Binding Lectin Associated with Protection against Tuberculosis Caused by Mycobacterium africanum but not by M. tuberculosis
- (2011) Thorsten Thye et al. PLoS One
- LocusZoom: regional visualization of genome-wide association scan results
- (2010) R. J. Pruim et al. BIOINFORMATICS
- Genome-wide association study identifies variants in the CFH region associated with host susceptibility to meningococcal disease
- (2010) Sonia Davila et al. NATURE GENETICS
- Genome-wide association analyses identifies a susceptibility locus for tuberculosis on chromosome 18q11.2
- (2010) Thorsten Thye et al. NATURE GENETICS
- The Sequence Alignment/Map format and SAMtools
- (2009) H. Li et al. BIOINFORMATICS
- Fast and accurate short read alignment with Burrows-Wheeler transform
- (2009) H. Li et al. BIOINFORMATICS
- Infection withMycobacterium tuberculosisBeijing Genotype Strains Is Associated with Polymorphisms inSLC11A1/NRAMP1in Indonesian Patients with Tuberculosis
- (2009) Reinout van Crevel et al. JOURNAL OF INFECTIOUS DISEASES
- The Influence of Host and Bacterial Genotype on the Development of Disseminated Disease with Mycobacterium tuberculosis
- (2008) Maxine Caws et al. PLoS Pathogens
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More