标题
The Lysosomal Trafficking Transmembrane Protein 106B Is Linked to Cell Death
作者
关键词
-
出版物
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 291, Issue 41, Pages 21448-21460
出版商
American Society for Biochemistry & Molecular Biology (ASBMB)
发表日期
2016-08-26
DOI
10.1074/jbc.m116.737171
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Increased expression of the frontotemporal dementia risk factor TMEM106B causes C9orf72-dependent alterations in lysosomes
- (2016) Johanna I. Busch et al. HUMAN MOLECULAR GENETICS
- Genetics of FTLD: overview and what else we can expect from genetic studies
- (2016) Cyril Pottier et al. JOURNAL OF NEUROCHEMISTRY
- Emerging mechanisms of molecular pathology in ALS
- (2015) Owen M. Peters et al. JOURNAL OF CLINICAL INVESTIGATION
- The cleavage pattern of TDP-43 determines its rate of clearance and cytotoxicity
- (2015) Quan Li et al. Nature Communications
- Common pathobiochemical hallmarks of progranulin-associated frontotemporal lobar degeneration and neuronal ceroid lipofuscinosis
- (2014) Julia K. Götzl et al. ACTA NEUROPATHOLOGICA
- TMEM106B is a genetic modifier of frontotemporal lobar degeneration with C9orf72 hexanucleotide repeat expansions
- (2014) Michael D. Gallagher et al. ACTA NEUROPATHOLOGICA
- TMEM106B protects C9ORF72 expansion carriers against frontotemporal dementia
- (2014) Marka van Blitterswijk et al. ACTA NEUROPATHOLOGICA
- Nuclear TDP-43 causes neuronal toxicity by escaping from the inhibitory regulation by hnRNPs
- (2014) Hiroaki Suzuki et al. HUMAN MOLECULAR GENETICS
- Regulated Intramembrane Proteolysis of the Frontotemporal Lobar Degeneration Risk Factor, TMEM106B, by Signal Peptide Peptidase-like 2a (SPPL2a)
- (2014) Owen A. Brady et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Lysosome size, motility and stress response regulated by fronto-temporal dementia modifier TMEM106B
- (2014) Massimiliano Stagi et al. MOLECULAR AND CELLULAR NEUROSCIENCE
- Defining the association of TMEM106B variants among frontotemporal lobar degeneration patients with GRN mutations and C9orf72 repeat expansions
- (2014) Serena Lattante et al. NEUROBIOLOGY OF AGING
- Possible involvement of lysosomal dysfunction in pathological changes of the brain in aged progranulin-deficient mice
- (2014) Yoshinori Tanaka et al. Acta Neuropathologica Communications
- The FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomes
- (2013) Benjamin M. Schwenk et al. EMBO JOURNAL
- Lysosomal cell death at a glance
- (2013) S. Aits et al. JOURNAL OF CELL SCIENCE
- TMEM106B p.T185S regulates TMEM106B protein levels: implications for frontotemporal dementia
- (2013) Alexandra M. Nicholson et al. JOURNAL OF NEUROCHEMISTRY
- State of play in amyotrophic lateral sclerosis genetics
- (2013) Alan E Renton et al. NATURE NEUROSCIENCE
- Converging Mechanisms in ALS and FTD: Disrupted RNA and Protein Homeostasis
- (2013) Shuo-Chien Ling et al. NEURON
- Current insights into the C9orf72 repeat expansion diseases of the FTLD/ALS spectrum
- (2013) Marc Cruts et al. TRENDS IN NEUROSCIENCES
- The genetics and neuropathology of frontotemporal lobar degeneration
- (2012) Anne Sieben et al. ACTA NEUROPATHOLOGICA
- The frontotemporal lobar degeneration risk factor, TMEM106B, regulates lysosomal morphology and function
- (2012) Owen A. Brady et al. HUMAN MOLECULAR GENETICS
- Membrane Orientation and Subcellular Localization of Transmembrane Protein 106B (TMEM106B), a Major Risk Factor for Frontotemporal Lobar Degeneration
- (2012) Christina M. Lang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- TMEM106B, the Risk Gene for Frontotemporal Dementia, Is Regulated by the microRNA-132/212 Cluster and Affects Progranulin Pathways
- (2012) A. S. Chen-Plotkin et al. JOURNAL OF NEUROSCIENCE
- Cellular ageing, increased mortality and FTLD-TDP-associated neuropathology in progranulin knockout mice
- (2012) Hans Wils et al. JOURNAL OF PATHOLOGY
- Klotho as a potential biomarker and therapy for acute kidney injury
- (2012) Ming-Chang Hu et al. Nature Reviews Nephrology
- Association of TMEM106B Gene Polymorphism With Age at Onset in Granulin Mutation Carriers and Plasma Granulin Protein Levels
- (2011) Carlos Cruchaga et al. ARCHIVES OF NEUROLOGY
- TMEM106B is associated with frontotemporal lobar degeneration in a clinically diagnosed patient cohort
- (2011) Julie van der Zee et al. BRAIN
- TDP-43-induced Death Is Associated with Altered Regulation of BIM and Bcl-xL and Attenuated by Caspase-mediated TDP-43 Cleavage
- (2011) Hiroaki Suzuki et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Molecular pathways of motor neuron injury in amyotrophic lateral sclerosis
- (2011) Laura Ferraiuolo et al. Nature Reviews Neurology
- Risk genotypes at TMEM106B are associated with cognitive impairment in amyotrophic lateral sclerosis
- (2010) Ryan Vass et al. ACTA NEUROPATHOLOGICA
- Inclusion body myopathy, Paget's disease of the bone and fronto-temporal dementia: a disorder of autophagy
- (2010) J. S. Ju et al. HUMAN MOLECULAR GENETICS
- Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions
- (2010) Vivianna M Van Deerlin et al. NATURE GENETICS
- TMEM106B regulates progranulin levels and the penetrance of FTLD in GRN mutation carriers
- (2010) N. Finch et al. NEUROLOGY
- Sortilin-Mediated Endocytosis Determines Levels of the Frontotemporal Dementia Protein, Progranulin
- (2010) Fenghua Hu et al. NEURON
- Oxidative Stress and Neurodegenerative Diseases: A Review of Upstream and Downstream Antioxidant Therapeutic Options
- (2009) Bayani Uttara et al. Current Neuropharmacology
- ALS-linked P56S-VAPB, an aggregated loss-of-function mutant of VAPB, predisposes motor neurons to ER stress-related death by inducing aggregation of co-expressed wild-type VAPB
- (2008) Hiroaki Suzuki et al. JOURNAL OF NEUROCHEMISTRY
- Lysosomal membrane permeabilization in cell death
- (2008) P Boya et al. ONCOGENE
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