Hypoxia‐inducible lncRNA MIR210HG interacting with OCT1 is involved in glioblastoma multiforme malignancy

Hypoxia-induced lncRNA PDIA3P1 promotes mesenchymal transition via sponging of miR-124-3p in glioma

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LncRNA MIR210HG promotes proliferation and invasion of non-small cell lung cancer by upregulating methylation of CACNA2D2 promoter via binding to DNMT1

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Ensembl 2019

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p53 and miR-210 regulated NeuroD2, a neuronal basic helix-loop-helix transcription factor, is downregulated in glioblastoma patients and functions as a tumor suppressor under hypoxic microenvironment

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Targeting Oct1 genomic function inhibits androgen receptor signaling and castration-resistant prostate cancer growth

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Long Noncoding RNA miR210HG as a Potential Biomarker for the Diagnosis of Glioma

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The Long Non-coding RNA HIF1A-AS2 Facilitates the Maintenance of Mesenchymal Glioblastoma Stem-like Cells in Hypoxic Niches

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Hypoxic signature of microRNAs in glioblastoma: insights from small RNA deep sequencing

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Exogenous IGFBP-2 promotes proliferation, invasion and chemoresistance to temozolomide in glioma cells via the integrin β1-ERK pathway

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Knockdown of miR-210 decreases hypoxic glioma stem cells stemness and radioresistance

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Extensive regulation of the non-coding transcriptome by hypoxia: role of HIF in releasing paused RNApol2

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Loss of corepressor PER2 under hypoxia up-regulates OCT1-mediated EMT gene expression and enhances tumor malignancy

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Long non-coding RNAs and cancer: a new frontier of translational research?

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Transcription Factor Oct1 Is a Somatic and Cancer Stem Cell Determinant

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Knock down of HIF-1α in glioma cells reduces migration in vitro and invasion in vivo and impairs their ability to form tumor spheres

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Long Noncoding RNA as Modular Scaffold of Histone Modification Complexes

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Intratumoral Hypoxic Gradient Drives Stem Cells Distribution and MGMT Expression in Glioblastoma

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Hypoxia-Inducible Factors Regulate Tumorigenic Capacity of Glioma Stem Cells

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Hypoxia-Inducible mir-210 Regulates Normoxic Gene Expression Involved in Tumor Initiation

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Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1

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EphA4 promotes cell proliferation and migration through a novel EphA4-FGFR1 signaling pathway in the human glioma U251 cell line

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