ALK-1 to ALK-5 ratio dictated by the Akt1-β-catenin pathway regulates TGFβ-induced endothelial-to-mesenchymal transition
出版年份 2020 全文链接
标题
ALK-1 to ALK-5 ratio dictated by the Akt1-β-catenin pathway regulates TGFβ-induced endothelial-to-mesenchymal transition
作者
关键词
ALK1, ALK5, TGFβ, EndMT, Akt1, β-Catenin
出版物
GENE
Volume 768, Issue -, Pages 145293
出版商
Elsevier BV
发表日期
2020-11-04
DOI
10.1016/j.gene.2020.145293
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- lncRNA H19 prevents endothelial–mesenchymal transition in diabetic retinopathy
- (2019) Anu A. Thomas et al. DIABETOLOGIA
- The Molecular Mechanism of Transforming Growth Factor-β Signaling for Intestinal Fibrosis: A Mini-Review
- (2019) Sun-Mi Yun et al. Frontiers in Pharmacology
- Endothelial stromelysin1 regulation by the forkhead box-O transcription factors is crucial in the exudative phase of acute lung injury
- (2019) Sandeep Artham et al. PHARMACOLOGICAL RESEARCH
- Endothelial-Mesenchymal Transition Drives Expression of CD44 Variant and xCT in Pulmonary Hypertension
- (2019) Sarasa Isobe et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Fibroblast growth factor signals regulate transforming growth factor‐β‐induced endothelial‐to‐myofibroblast transition of tumor endothelial cells via Elk1
- (2019) Yuichi Akatsu et al. Molecular Oncology
- Pharmacological inhibition of β-catenin prevents EndMT in vitro and vascular remodeling in vivo resulting from endothelial Akt1 suppression
- (2019) Harika Sabbineni et al. BIOCHEMICAL PHARMACOLOGY
- Endothelial–mesenchymal transition in atherosclerosis
- (2018) Celine Souilhol et al. CARDIOVASCULAR RESEARCH
- NLRP3 Inflammasome Activation Contributes to Mechanical Stretch–Induced Endothelial-Mesenchymal Transition and Pulmonary Fibrosis
- (2018) Zhou Lv et al. CRITICAL CARE MEDICINE
- Isoform-specific effects of transforming growth factor β on endothelial-to-mesenchymal transition
- (2018) Harika Sabbineni et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Reassessing endothelial-to-mesenchymal transition in cardiovascular diseases
- (2018) Yan Li et al. Nature Reviews Cardiology
- miR-449a induces EndMT, promotes the development of atherosclerosis by targeting the interaction between AdipoR2 and E-cadherin in Lipid Rafts
- (2018) Lei Jiang et al. BIOMEDICINE & PHARMACOTHERAPY
- Tumour-vasculature development via endothelial-to-mesenchymal transition after radiotherapy controls CD44v6+ cancer cell and macrophage polarization
- (2018) Seo-Hyun Choi et al. Nature Communications
- Modulation of long-term endothelial-barrier integrity is conditional to the cross-talk between Akt and Src signaling
- (2017) Fei Gao et al. JOURNAL OF CELLULAR PHYSIOLOGY
- TGF-β promotes PI3K-AKT signaling and prostate cancer cell migration through the TRAF6-mediated ubiquitylation of p85α
- (2017) Anahita Hamidi et al. Science Signaling
- TGF-β promotes PI3K-AKT signaling and prostate cancer cell migration through the TRAF6-mediated ubiquitylation of p85α
- (2017) Anahita Hamidi et al. Science Signaling
- Akt1 promotes stimuli-induced endothelial-barrier protection through FoxO-mediated tight-junction protein turnover
- (2016) Fei Gao et al. CELLULAR AND MOLECULAR LIFE SCIENCES
- Signaling Receptors for TGF-β Family Members
- (2016) Carl-Henrik Heldin et al. Cold Spring Harbor Perspectives in Biology
- The Akt inhibitor, triciribine, ameliorates chronic hypoxia-induced vascular pruning and TGFβ-induced pulmonary fibrosis
- (2015) Maha Abdalla et al. BRITISH JOURNAL OF PHARMACOLOGY
- ALK5 and ALK1 Play Antagonistic Roles in Transforming Growth Factor -Induced Podosome Formation in Aortic Endothelial Cells
- (2014) F. Curado et al. MOLECULAR AND CELLULAR BIOLOGY
- Inflammatory Stress Exacerbates the Progression of Cardiac Fibrosis in High-Fat-Fed Apolipoprotein E Knockout Mice via Endothelial-Mesenchymal Transition
- (2013) Kun Ling Ma et al. International Journal of Medical Sciences
- Signaling interplay between transforming growth factor-β receptor and PI3K/AKT pathways in cancer
- (2013) Long Zhang et al. TRENDS IN BIOCHEMICAL SCIENCES
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started